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Circulation: Amal Alabbadi Sukainah Alalwi Fatimha Almarhoon Khadijah Almubarak

This document discusses the management of different types of non-hemorrhagic shock, including anaphylactic, cardiogenic, tension pneumothorax, septic, and shock caused by pulmonary embolism or cardiac tamponade. The management of each type depends on identifying and treating the underlying cause, with goals of improving tissue perfusion and oxygen delivery through measures like fluid resuscitation, vasopressors, inotropes, antibiotics, needle decompression, or surgical drainage.

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0% found this document useful (0 votes)
58 views33 pages

Circulation: Amal Alabbadi Sukainah Alalwi Fatimha Almarhoon Khadijah Almubarak

This document discusses the management of different types of non-hemorrhagic shock, including anaphylactic, cardiogenic, tension pneumothorax, septic, and shock caused by pulmonary embolism or cardiac tamponade. The management of each type depends on identifying and treating the underlying cause, with goals of improving tissue perfusion and oxygen delivery through measures like fluid resuscitation, vasopressors, inotropes, antibiotics, needle decompression, or surgical drainage.

Uploaded by

ali
Copyright
© © All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
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Download as PPTX, PDF, TXT or read online on Scribd

Circulation

Amal Alabbadi
Sukainah Alalwi
Fatimha Almarhoon
Khadijah almubarak
Objective

[Link] understand the causes of shock


2. To recognize shock utilizing the physical examination
3. To review the management of different types of shock
What are the causes of shock?
Amal Alabbadi
2160002144
Definition of shock:
An abnormality of the circulatory system
causing inadequate tissue perfusion which,
if not corrected, will result in → cell death.
Causes of shock

Hypovolemic shock Cardiogenic Obstructive shock Distributive Dissociative shock


shock shock
Causes of shock

Hypovolemic shock Cardiogenic Obstructive shock Distributive Dissociative shock


shock shock

Hemorrhagic Non-hemorrhagic
shock hypovolemic
shock

Inadequate intake
Excessive output
Causes of shock

Hypovolemic shock Cardiogenic Obstructive shock Distributive Dissociative shock


shock shock

Primary cardiac disorder

 Pump failure from myocardial injury or dysfunction: Myocarditis,


cardiomyopathy, ischemia, infarct.
 Arrhythmias
 Valvular disease
 Ventricular septal defect
Causes of shock

Hypovolemic shock Cardiogenic Obstructive shock Distributive Dissociative shock


shock shock

Extra-cardiac obstruction

Cardiac tamponade
Pulmonary embolism
Tension pneumothorax
Causes of shock

Hypovolemic shock Cardiogenic Obstructive shock Distributive Dissociative shock


shock shock

Sepsis and Anaphylaxis Neurogenic shock


SIRS

Cascade of cytokines Inflammatory mediators: Acute spinal


& other immune and Histamine injury
inflammatory Leukotriene C4
modulators Prostaglandin D2
Causes of shock

Hypovolemic shock Cardiogenic Obstructive shock Distributive Dissociative shock


shock shock

CO poisoning
Cyanide poisoning
Physical examination of shock.
Sukainah Al-alawi
2160005104
Recognizing shock.

• General appearance.
• Vital Signs.
• Mental status.
• Skin changes.
• CV: JVP, heart sounds.
Physical examination:

Pulse: Respiratory Rate: Blood pressure:


Tachycardia Tachypnea Hypotension

Mental status: JVP: Heart Sounds:


Early: Agitation. Low: Hypovolemia, Muffled heart sounds
Late: Sepsis. may be noted in
High: LV Failure, Right cardiac tamponade.
Obtundation.
Heart Problems.
Orthostatic Vital Signs:

1. Change in pulse and blood


pressure as a patient is moved
from a supine to sitting or erect
position.
2. No general agreement to what
changes constitute a ‘positive
response’.
3. The test is insensitive and
nonspecific.
4. Never be performed in a
potentially unstable patient.
Continue…

Skin Capillary Blanch Test: The exception to this


rule is in vasogenic
1. Cool and pale early on.
1. Nail bed takes >2 shock.
2. Peripheral cyanosis
seconds to ‘pink up’.
may appear later.
2. If acute blood loss
excess 15% of total Why ?
blood volume.
Management of hemorrhagic shock
Fatimha Almarhoon
2160002959.
HEMORRHAGIC SHOCK
A 23 year old man got into a car
accident and he was thrown 22
feet away.
He was taken to the ED and was
unconscious and pale and fresh
blood was flowing from a
laceration in his right groin. His
vitals:
T 37.0
HR 150 bpm
BP 90/40
RR 24
O2 Sat 78

What is your next step in management?


General management
ABC
AIRWAY, BREATHING, O2
SAT, CARDIAC MONITOR
01 02 INTRAVENOUS ACCESS
CROSS MATCH IF
HEMORRHAGE SUSPECTED

CONTROL EXTERNAL BLEEDING


BY APPLYING PRESSURE
03 04 FOLEY CATETHER
MONITOR URINE OUTPUT

ONGOING ASSESSMENT
BP, PR, RR,SKIN, LOC
05 06 INVASIVE
MONITORING
ATRIAL LINE
Specific management

Volume resuscitation Blood products Hemorrhagic control

• NS or Ringer’s lactate • Blood products if no • Address or control source


boluses response to two fluid of bleeding
• 1–2 L in adults ■ 10– boluses, ongoing
20 cc/kg in neonates, hemorrhage, or if
infants, and young impending
children cardiovascular
collapse
• Two units PRBC in
adults ■ 10–15 mL/kg
PRBC in neonates,
infants
• Platelet
• O- blood
Specific management
1. Restore circulating blood volume
Treatment for hemorrhagic shock consisted of rapidly and aggressive infusing several
liters of isotonic crystalloid by using:
• At least two short, large bore catheters
• Pressure infusion devices

2. Access
• Peripheral access (preferred):
Sites: forearm, antecubital, saphenous vein cutdown
• Central access:
Sites: femoral vein, internal jugular vein, subclavian vein

3. Consultation
Management Of
Non-hemorrhagic
Shock
Khadijaa Ahmed Almubarak
2160003996
NON-HEMORRHAGIC SHOCK

A 45-year-old female is brought to


the ED after collapsing at home
shortly after sustaining a bee sting
to her left hand, the patient is
unresponsive & without palpable
pulse, but breathing.

Her vital signs: BP: 60/40, HR:


130s, RR: 20
Anaphylactic shock

Management:
 Airway management
 IM Epinephrine
 Fluid management: crystalloid boluses via large-bore IVs
 Histamine blockade: Diphenhydramine (H1) , Cimetidine (H2)
 Corticosteroid to reduce inflammation
NON-HEMORRHAGIC SHOCK

A 22-year-old previously healthy


male presents to the ED
complaining of flulike symptoms
and progressive shortness of
breath

He appears ill with BP 80/50, HR


120, RR 30 and T 38.0ºC. He has
evidence of poor perfusion, and
jugular venous distention.
Cardiogenic Shock

Management:
 Will depend on the underlying cause
 The goal is to improve the myocardial contractility & pump function
 Ensure adequate ventilation & oxygenation
 Intropes for depressed LV function: Dobutamine
 Other options include emergency angioplasty
NON-HEMORRHAGIC SHOCK

A 19-year-old male presents to the


ED Stabbed in his anterior chest,
looks gasping, intoxicated with
sever SOB

BP 88/42, HR 110, RR 30
Tension pneumothorax

Management:
 Ensure adequate ventilation & oxygenation
 Needle decompression: 14- or 16-gauge needle in the 2 nd or 3rd intercostal
midclavicular line
 Chest tube: in the 4th intercostal space anterior axillary line (skin incision 1
ICS below)
Other Causes

Septic shock
Cardiac temponade intavenous crystalloid,
Intravenous crystaloid, definitive therapy (AB,
pericardiocentesis surgical drainage)

Massive PE
Arrhythmias
Intravenous crystalloid,
Specific anti-arrhythmic
inotropes, thrombolysis or
therapy
surgery
Non-Hemorrhagic Shock

Anaphylactic Cardiac
Arrhythmias
Shock Tamponade

Cardiogenic Tension
Septic Shock
Shock Pneumothorax

Massive PE
REFERENCES

1. Blok B, Cheung D, Platts-Mills T. First aid for the emergency medicine


boards.
2. Helman A, Pencinar R. The ABCs of emergency medicine. 7th ed. 2008.
Thank you
Any questions?

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