RESPIRATORY SYSTEM
Anatomy and physiology
The upper respiratory tract, known as the upper airway, warms and filters inspired air so that the lower respiratory tract
(the lungs) can accomplish gas exchange.
Gas exchange involves delivering oxygen to the tissues through the bloodstream and expelling waste gases, such as carbon
dioxide, during expiration.
The respiratory system works in concert with them cardiovascular system; the respiratory system is responsible for
ventilation and diffusion, and the cardiovascular system is responsible for perfusion.
Function of the Respiratory System
1. Oxygen Transport
Oxygen is supplied to, and removed from cell by the way of circulating blood.
Oxygen diffuses from the capillary wall to the interstitial fluid.
The movement of the carbon dioxide occurs by diffusion in the opposite direction –from cell to blood
2. Respiration
Movement of air in and out of the airway (ventilation) continually replenishes the oxygen and removes carbon dioxide
from the airway and lungs.
3. Ventilation
During inspiration, air flow from the environment into the trachea, bronchi, bronchioles and alveoli. During expiration,
alveolar gas travels the same route in reverse.
Mechanism of ventilation includes:
a. Air pressure variance – air flow from a region of higher pressure to a region of lower pressure.
b. Airway Resistance – is determined chiefly by the radius or size of the airway through which air is flowing. With
increase resistance, greater than the normal respiratory effort is required to achieve normal levels of ventilation
c. Compliance- refers to which the lungs expand and indicates the relationship between the volume and the pressure
of the lungs
4. Regulation of Acid-Base Balance
Insufficient ventilation causes hypercapnia, a respiratory acidemia causes retention of excessive amount of CO2.
Hypocapnia, a respiratory alkalemia due to the low amounts of CO2, in the blood
The effectiveness of ventilation is best measured by the PCO2 in the arterial blood (PaCO2)
THE RESPIRATORY PROCESS
1. The diaphragm descends into the abdominal cavity during inspiration causing (-) pressure in the lungs.
2. The (-) pressure draws the air from the area of greater pressure (THE ATMOSPHERE) into an area of lesser pressure (THE
LUNGS)
3. In the lungs, air passes thru the terminal bronchioles into the alveoli to oxygenate the body tissues
4. At the end of inspiration, the diaphragm & intercostal muscles relax & the lungs recoil
5. As the lungs recoil, pressure within the lungs becomes greater than atmospheric pressure, causing the air which now
contains the cellular waste products of CO2 & H2O to move from the alveoli in the lungs to the atmosphere
6. Expiration is a passive process
Control of Respiration
1. Activity of the respiratory muscles is regulated by nerve impulses transmitted by the brain thru the Phrenic & Intercostal
nerves.
2. Neural centers that control breathing are located in the medulla oblongata and the pons
3. Eupnea – normal breathing
Normal breath sounds
1. Bronchial
Loud and high pitched w/ hollow quality.
Expiration lasts longer than inspiration.
Best heard over the trachea
Created by air moving through the trachea close to chest wall.
2. Bronchovesicular
Blowing sounds that are moderate in pitch and intensity. Inspiration is equal to expiration.
Best heard posteriorly between scapulas & anteriorly over bronchioles lateral to sternum at first & second intercostal
spaces.
Created by air moving to large airways.
Abnormal breath sounds
1. Stridor
A loud, high-pitched crowing sound that is heard, usually w/o a stethoscope, during inspiration.
Stridor caused by an obstruction in the upper airway requires immediate attention.
2. Rhonchi (also called gurgles)
Low-pitched, snoring sounds that occur when the pt. exhales, although they may also be heard when the pt. inhales.
Usually change or disappear w/ coughing
Sounds occur as a result of air passing through fluid-filled, narrow passages, diseases where there is increased mucus
production such as pneumonia, bronchitis, or bronchiectasis.
3. Crackles ( Rales )
Soft, high pitched discontinuous popping sounds that occur during inspiration
Can be produced by rubbing a lock of hair between the thumb and finger close to the ear.
Fluid in the airways
Obstructive disease in early inspiration, Bronchitis and pneumonia, CHF
4. Wheeze
deep, low-pitched sounds heard during exhalation
due to narrowed tracheobronchial passages from secretions
Continuous, musical, high-pitched, whistle - like sounds heard during inspiration and exhalation
narrow bronchioles, associated with bronchospasm, asthma and buildup of secretions
4. Friction Rub
Like 2 pieces of rubber rubbed together, inspiration and exhalation
Inflammation and loss of fluid in the pleural space
Associated with pleurisy, pneumonia, or pleural infarct.
RISK FACTORS FOR RESPIRATORY DISEASE
1. Smoking
2. Tobacco
3. Allergies
4. Frequent respiratory illnesses
5. Chest injury
6. Surgery
7. Chemicals & environmental pollutants
8. Family history of infectious disease
9. Geographic residence
10. Travel to foreign countries
DIAGNOSTIC TESTS
1. CHEST X-RAY (CXR) FILM (RADIOGRAPH)
- Information on the anatomic location & appearance
PRE-PROCEDURE NURSING CARE
1. remove jewelry band other metal object
2. inhale and hold breath
3. assess for pregnancy
2. SPUTUM SPECIMEN
- Expectoration or tracheal suctioning
- identify organisms or abnormal cells
PRE-PROCEDURE NURSING CARE
1. Determine purpose
2. Early morning specimen
3. 15 ml
4. Rinse the mouth with water prior to collection
5. Deep breaths then cough
6. Collect specimen before antibiotics
3. SUCTIONING PROCEDURE IN OBTAINING SPUTUM SPECIMEN
1. Aseptic technique
2. Hyperoxygenate before and after
3. Lubricate catheter with sterile water
4. Tracheal suctioning: 4 inches
5. Nasotracheal suctioning: insert to induce cough
6. Suction intermittently for 10 to 15 sec
7. Rotate and withdraw
8. Hyperoxygenate & deep breaths
4. BRONCHOSCOPY
PRE-PROCEDURE NURSING CARE
1. Informed consent
2. NPO prior
3. Coagulation studies
4. Remove dentures or eyeglasses
5. Prepare suction
6. Sedatives
• Resuscitation equipment available
POST-PROCEDURE NURSING CARE
1. V/S
2. Ý Fowler’s
3. CHECK GAG REFLEX
4. NPO
5. Monitor for bloody sputum
6. Monitor respiration
7. Monitor for complications
Notify the MD if complications occur
5. PULMONARY ANGIOGRAPHY
insertion of a fluoroscopy via the antecubital or femoral vein into the pulmonary artery
it involves iodine or radiopaque or contrast material
PRE-PROCEDURE NURSING CARE
1. Secure consent
2. Assess for allergy to seafood
3. Remain still during procedure
POST-PROCEDURE NURSING CARE
1. No BP for 24 hours in the affected extremity
2. Monitor neurovascular status
3. Assess bleeding
4. Monitor dye reaction
6. THORACENTESIS
PRE-PROCEDURE NURSING CARE
1. CXR or U/S prior to the procedure
2. Upright
3. Do not to cough, breathe deeply, or move during the procedure
POST-PROCEDURE NURSING CARE
1. Monitor respiratory status
2. Pressure dressing
3. Assess site for bleeding and crepitus
7. LUNG BIOPSY
PRE-PROCEDURE NURSING CARE
1. Local anesthetic
2. Pressure during insertion and aspiration
3. Administer analgesics & sedatives as Rx
POST-PROCEDURE NURSING CARE
1. Pressure dressing
2. Monitor for bleeding
3. Monitor for respiratory distress
4. Monitor for complications
5. Prepare for CXR
8. VENTILATION PERFUSION LUNG SCAN - determines the patency of the pulmonary airways
PRE-PROCEDURE NURSING CARE •
• Assess for allergy to seafood
• Remove jewelry
• IV access
• Administer sedation
• Emergency resuscitation equipment
POST-PROCEDURE NURSING CARE
Handle secretions carefully for 24 hours
9. SKIN TESTS
PRE-PROCEDURE NURSING CARE
• Determine hypersensitivity or previous reactions to skin tests
PROCEDURE
1. Should be off excessive body hair & dermatitis
2. Circle, document the date, time and test site
POST-PROCEDURE NURSING CARE
1. Do not to scratch
2. Do not wash
3. Assess for induration (hard swelling), erythema and vesiculation (small blister-like elevations)
10. PULSE OXIMETRY
- NORMAL VALUE: 95% - 100%
PROCEDURE
1. A sensor is placed: finger, toe, nose, earlobe or forehead
2. Don’t select an extremity with an impediment to blood flow
3. Lower than 91% - immediate treatment
4. Lower than 85% - hypooxygenation
5. Lower than 70% - life-threatening situation
11. CHEST PHYSIOTHERAPY (CPT)
NURSING CARE
1. Morning upon arising
2. 1 hr before meals or 2-3 hrs after meals
3. Stop if pain occurs
4. Mouth care
CONTRAINDICATIONS OF CHESTPHYSIOTHERAPY (CPT)
1. Ý respiratory distress
2. Hx of fractures
3. Chest incisions
12. POSTURAL DRAINAGE - use of the gravity
1. A.M. upon arising
2. 1 hour before meals, 2-3 hours after meals
3. Stop if cyanosis or exhaustion occurs
4. Maintain position 5-20 minutes after
5. Mouth care
6. Unstable V/S
7. Increased ICP
13. INCENTIVE SPIROMETRY
1. Lips should seal the mouth piece
2. Inspire deeply
3. Hold inspiration
4. Forcefully exhale
5. Avoid the use at mealtimes
OXYGEN (O2) ADMINSITRATION
1. NASAL CANNULA (NASAL PRONGS)
- 1-6L/min
- 24% to 44%
FI02 DELIVERED VIA NASAL CANNULA
24% at 1 L/min
28% at 2 L/min
32% at 3 L/min
36% at 4 L/min
40% at 4 L/min
44% at 5 L/min
NURSING CARE
1. Humidification
2. Monitor humidifier
3. Assess RR
4. Assess mucosa
5. Assess skin integrity
6. Water-soluble jelly
Advantages
1. Most commonly used oxygen delivery device
2. Safe and simple; comfortable and easily tolerated
3. Effective for low oxygen concentration
4. Does not impede eating or speaking.
5. Low flow 24% - 44%
Disadvantages
1. it can be easily be dislodge and can cause dryness of the nasal mucosa and headache if flow rate exceeds 6 L/min
2. Can’t deliver concentration higher than 44%
3. Can’t be used in complete nasal obstruction
2. SIMPLE FACE MASK
- 40% - 60%
- short term O2 therapy
- minimal flow rate of 5 L/min
NURSING CARE
1. Fitting mask
2. Skin care
3. Monitor for aspiration
4. Emotional support
5. Claustrophobic
Advantages
1. Simple mask is used when an increased delivery of oxygen is needed for short period (less than 12 hours)
2. The mask should fit closely to the face to deliver this higher concentration of oxygen effectively.
Disadvantages
1. Hot and confining; may irritate patient’s skin
2. Tight seal which may cause discomfort
3. Interfere with talking and eating
4. Impractical for long-term therapy because of imprecision
3. PARTIAL REBREATHER MASK
- 70% - 90%
- 6 – 15 L/min
- rebreathes 1/3 of the exhaled TV
NURSING CARE
• Reservoir should not twist or kink
• Reservoir inflated 2/3 full on inspiration
Advantages
1. Opening in the mask allow patient to inhale room air if oxygen source fails
2. The patient rebreathes about 1/3 of the expired air from the reservior bag.
Disadvantages
1. Eating and talking is difficult
2. Tight seal is required
3. Potential for skin breakdown
4. NON-REBREATHER MASK
- Ý 90%
- Use in deteriorating respiratory status
NURSING CARE
1. Remove mucus or saliva
2. Assess client
3. Valve & flaps functional
4. Valves open during expiration
5. Close during inspiration
6. Monitor kinks & twisting
5. VENTURI MASK
1. accurate O2 inhalation
2. Adapter contains holes
3. Adapter allows selection of the amount of O2 desired
4. 24% to 55%
5. 4-10L/min
NURSING CARE
1. Monitor closely
2. Mask fits snugly
3. Tubing is free of kinks
4. Monitor mucous membranes
Disadvantage
Hot and confining; may irritate patient’s skin
Tight seal which may cause discomfort
Interfere with talking and eating
O2 concentration may be altered if mask is loosely fit, tubing kinks, o2 intake ports become blocked, flow is insufficient,
or patient is hyperneic
6. FACE TENT - useful for client with facial trauma or burn
7. AEROSOL MASK - used for the client who has thick secretions
8. TRACHEOSTOMY COLLAR OR T-PIECE
- For high humidity & the desired O2 to the client with a endotracheal or tracheostomy
FACE TENT, AEROSOL MASK, TRACHEOSTOMY COLLAR & T- PIECE
NURSING CARE
1. Nasal cannula during meals
2. Empty condensation
3. Monitor water in the canister
4. Exhalation port in the T-piece always open
9. OXYGEN TENT
1. Is a bendable piece of clear plastic held over your child's bed or crib by a frame.
2. The plastic is then tucked under the mattress. It may also be called a croup, mist, or Ohio tent.
3. Oxygen or regular air is blown into the tent.
4. Oxygen tent allows for delivery for between 30%-50% humidified oxygen
5. Maintain temperature at 17.8°C-21.2°C
6. Secure the canopy by tucking in all sides and maintain closure whenever possible to prevent oxygen leak at the bottom of
tent.
7. Avoid use of friction-type toys or battery operated device when oxygen is in use
8. Check dampness of clothes to prevent chilling.
ARTIFICIAL AIRWAY
A. Endotracheal Tube
Purpose:
1. Tracheal Suctioning
2. Positive Pressure Breathing
Nursing. Care:
1. Humidify air
2. Suction PRN
3. NGT
4. Promote Communication
5. Confirm placement
6. Monitor the cuff
B. TRACHEOSTOMY TUBE
Purpose: Same As Et
Types:
1. Plastic
2. Metal
Parts:
1. Outer Cannula
2. Inner Canula
3. Obsturator
Nursing. Care:
1. Asepsis
2. No sedative
3. Suction PRN
4. Hemostats
5. NGT, TPN & Oral nutrition
6. Wash the stoma
7. Tub bath
8. Avoid swimming
9. Weaning
RESPIRATORY DISORDERS
1. CHEST INJURIES
A. RIB FRACTURE - results from blunt chest trauma
NURSING CARE
1. Note that ribs unite spontaneously
2. ÝFowler’s
3. Pain medications
4. Monitor for respiratory distress
5. Instruct the client to self-splinting
6. Prepare for possible intercostal nerve block
B. FLAIL CHEST
ASSESSMENT
1. Paradoxical respirations
2. Severe chest pain
3. dyspnea
4. Cyanosis
5. Tachycardia
6. Hypotension
7. Tachypnea
8. Diminished breath sounds
NURSING CARE
1. ÝFowler’s
2. Humidified O2
3. Monitor respiratory distress
4. Coughing & deep breathing
5. Pain meds
6. Bed rest
7. Positive end-expiratory pressure ( PEEP ) for severe
C. PULMONARY CONTUSION - intra-alveolar hemorrhage resulting to ACUTE RESPIRATORY DISTRESS SYNDROME (ARDS)
ASSESSMENT
1. Dyspnea
2. Hypoxemia
3. Ý bronchial secretions
4. Hemoptysis
5. Restlessness
6. Decreased breath sounds
7. Rales and wheezes
NURSING CARE
Maintain airway
ÝFowler’s
O2 as Rx
Monitor respiratory distress
Maintain bed rest
PEEP
D. PNEUMOTHORAX
accumulation of atmospheric air in the pleural space
may lead to lung collapse
KINDS
1. SPONTANEOUS PNEUMOTHORAX
2. OPEN PNEUMOTHORAX
3. TENSION PNEUMOTHORAX
Dyspnea
Tachycardia
Tachypnea
Sharp chest pain
Absent breath sounds
ß chest expansion unilaterally
Cyanosis
Hypotension
Sucking sound
Tracheal deviation
NURSING CARE
1. Apply dressing over an open chest wound
2. O2 as Rx
3. Ý Fowler’s
4. Chest tube
CHEST TUBE DRAINAGE SYSTEM
1. returns (-) pressure to the intra-pleural space
2. remove abnormal accumulation of air & fluids
3. serves as lungs while healing is going on
A. COLLECTION CHAMBER
B. WATER SEAL CHAMBER
C. SUCTION CONTROL CHAMBER
Principles:
a. Gravity
b. Suction
c. Waterseal
NURSING CARE
1. Occlusive dressing
2. A CXR assesses the position of the tube & determines re-expansion
3. Assess respiratory status
4. Drainage system below the chest
5. Ensure secure connections
6. Coughing &DBE
7. Change position q 2
8. Do not strip
CHRONIC OBSTRUCTIVE PULMONARY DISEASE (COPD)
a group of diseases that includes - -EMPHYSEMA, ASTHMA, BRONCHIECTASIS CHRONIC BRONCHITIS
CHRONIC BRONCHITIS
Bronchial Inflammation Þ Ý mucus Þ ß cilia Þ respiratory acidosis
Causes:
Smoking
Pollution
Allergens
Assessment:
1. Chronic Cough
2. Blue Bloater:
3. Cyanotic
4. Edema
5. Chronic cough
6. Exertional dyspnea
7. ÝRR
8. Hypoxia
9. Polycythemia- ÝRBC
10. Hypercapnia
11. Cor pulmonale-RVH & dilatation
12. Resp. acidosis
13. Incidence in heavy cigarette smokers
EMPHYSEMA
Destruction and Overdistension of the Alveoli
ß
Air Trapping
ß
Respi. Acidosis
CAUSES:
1. Smoking, Pollution and Allergens
2. ß alpha-antitrypsin – causes expansion of the alveolI - strengthens the walls of the alveoli (blebs)
Assessment:
pink puffer:
1. Mucus secretions
2. Speaks in short & jerky sentence
3. Coughing
4. Anxiety
5. Orthopneic position
6. Frequently develop URTI
7. Barrelled chest
8. Prolonged expiratory time
9. SOB
10. Digital clubbing
11. Wheezing
BRONCHIECSTASIS
Permanent dilation & distension of the bronchi; may lead to Ý mucus production Þ respiratory Acidosis
The most common symptoms of bronchiectasis include: a persistent cough that usually brings up phlegm (sputum)
CAUSES:
1. Infection
2. Atelectasis
3. Aspiration
ASSESSMENT:
1. Mucupurelent mucus
2. Dyspnea
3. Fever
4. Orthopneic position
5. Anxiety
ASTHMA
- Characterized by recurring episodes of paroxysmal dyspnea, wheezing on inspiration/expiration caused by constriction of
the bronchi and viscous mucus secretions.
TYPES:
1. Extrinsic
2. Intrinsic – asthma w/ physiological cause
3. STATUS ASTMATICUS– severe form of constriction & inflammation despite treatment; may lead to respiratory or cardiac
failure.
ASSESSMENT:
1. Exertional Dyspnea
2. Barrelled chest
3. Hyperesonance
4. Spontaneous pneumothorax
Pharmacologic Therapy for COPD and Asthma
1. Bronchodilators:
Xanthines, aminophyline, theophyline
2. Adrenergics:
a. Isoproterenol(Isuprel),
b. Terbutaline,(Brethine),
c. Metaproterenol(Aluputent)
3. Expectorants: Guaifenessin(Robitusin)
4. Mucolytics: Acetylcysteine(Mucomyst)
5. Steroids: Prednisone
6. Propylaxis (anti-allergy): Cromolyn Na(Intal)
NURSING CARE
V/S
ß O2 conc. ( 2L)as Rx
Monitor pulse oximetry
Respiratory & chest physiotherapy
Pursed-lip breathing
Record the color, amount & consistency of sputum
Suction
Daily wt.
Small, frequent feedings
Ý calorie & CHON diet with supplements
Encourage fluids
Ý Fowler’s
Stop Smoking
Activity as tolerated
Avoid powerful odors
PNEUMONIA- is an inflammation of the lung parenchyma caused by various organisms including bacteria, mycobacteria,
clamydia, mycoplasma, fungi, parasites, and viruses.
ASSESSMENT
1. Grade fever
2. Chills
3. Chest pain
4. Grating sound
5. Rusty Sputum
6. Rales or crackles on auscultation
7. Dullness or hyperesonance
8. Dx test:
x-ray
gram-staining
sputum culture & sensitivity
NURSING CARE for PNEUMONIA
1. Fluids
2. Chest Physiotherapy
3. Chest splinting
4. Incentive Spirometer
5. calorie & CHON diet
6. Small frequent meals
7. Rest & activity as tolerated
8. Antibiotics as Rx
a. Azthromycin
b. Clarythromycin
c. Doxycycline
d. Fluoroquinolone
e. Ceftrioxone
f. inezolid (Zyvox)
g. Nafcelline
h. clyndamycin
LUNG CANCER
- Tumor in the Bronchial Epithelium; men 40 & Ý
TYPES:
1. Epidermoid/Squamous:
2. Adenocarcinoma
3. Small cell(Oat cell)
4. Large cell
CAUSES:
1. Genetics
2. Carcinogens
3. Infection
4. Smoking
ASSESSMENT:
1. Respiratory Pattern Changes
2. Hemoptysis
3. Dyspnea
4. Chest Pain
5. Fatigue
6. Anorexia
7. Persistent Dry Cough
Dx Test:
Sputum cytology
Lung biopsy
Bronchoscopy
NSG. RESPONSIBILITIES:
1. Early detection
2. Radiation – Cobalt
3. Chemotheraphy – does not distinguish normal from abnormal
4. Surgery – tx of choice
a. Pneumonectomy
b. Lobectomy
* Segment resection
* Wedge resection
PULMONARY TUBERCULOSIS
Highly communicable disease caused by a gram + acid-fast bacili (mycobacterium tuberculosis)
Causes/ Ý Risk groups:
1. Imunosuppression
2. Overcrowding
3. 3rd world country
4. Children ß5 yrs.old
5. Alcoholics
6. Smoking
ASSESSMENT:
1. Asymptomatic
2. Anorexia
3. Wt. Loss
4. Fatigue
5. Low grade P.M. fever
6. Night sweats
7. Sputum – yellow green
8. Hemoptysis
9. Chest pain
10. Ý tactile fremitus
Classifications of TB
Class 0: no exposure; no infection
Class 1: exposure; no evidence of infection
Class 2: latent infection; no disease (eg, positive PPD reaction but no clinical evidence of active TB)
Class 3: disease; clinically active
Class 4: disease; not clinically active
Class 5: suspected disease; diagnosis pending
Diagnostic Test:
1. Sputum test
2. Sputum Culture – TOC
3. Tuberculin test – Check for the presence of antibodies due to exposure
a. Mantoux test
b. Multiple puncture test (Tine or Monovac)
Nursing Care
1. Chemoprophylaxis – only indicated in primary infection
2. Multi-drug therapy
R-ifampicin
I-NH
P-yrazinamid
E-tambutol
S-treptomycin
PLEURAL EFFUSION
collection of fluid in the pleural space
ASSESSMENT
a. Sharp pleuritic pain
b. Dyspnea
c. Dry non-productive cough
d. Tachycardia
e. Ý temperature
f. ß breath sounds
g. CXR shows pleural effusion & a mediastinal shift away from the fluid
NURSING CARE
a. Identify & treat underlying cause
b. Monitor breath sounds
c. Monitor pulse oximetry
d. ÝFowler’s
e. Coughing & DBE
f. Thoracentesis
g. If pleural effusion is recurrent, prepare the client for pleurectomy or pleurodesis
PLEURECTOMY- surgically stripping the parietal pleura
PLEURODESIS - involves instillation of a sclerosing substance into the pleural space via a thoracotomy tube
EMPYEMA- pus within the pleural cavity; fluid is thick, opaque & foul smelling
ASSESSMENT
Fever & chills
Chest pain
Cough
Dyspnea
Anorexia & wt. loss
Malaise
Night sweats
Diminished chest wall movement on the affected side
Pleural exudate on chest CXR
NURSING CARE
Monitor breath sounds
ÝFowler’s
Coughing & DBE
Antibiotics as Rx
Chest splinting
PLEURISY- inflammation of the visceral & parietal membranes, may be caused by pulmonary infarction or pneumonia
ASSESSMENT
Sharp pleuritic pain
Dyspnea
Dry non-productive cough
Tachycardia
NURSING CARE
Identify & treat cause
Monitor lung sounds
Analgesics as Rx
Apply hot & cold applications as Rx
Coughing & DBE
Instruct the client to lie on affected side to splint chest
PULMONARY EMBOLISM
1. Dislodgement of thrombus to the pulmonary artery
2. Caused by thrombus & pulmonary emboli
3. Other risk factors: deep vein thrombosis, immobilization, surgery, obesity, pregnancy, CHF, advanced age, prior history of
thromboembolism
ASSESSMENT
Dyspnea
Chest pain
Tachypnea & tachycardia
Hypotension
Shallow respirations
Rales on auscultation
Cough
Blood-tinged sputum
Distended neck veins
Cyanosis
NURSING CARE
O2 as Rx
Ý Fowler’s
Maintain bed rest
Incentive spirometry as Rx
Pulse oximetry
Prepare for intubation & mechanical ventilation
IV heparin (bolus)
Warfarin (Coumadin)
Monitor PT & PTT closely
Prepare the client for embolectomy, vein ligation, or insertion of an umbrella filter as Rx
CARBON MONOXIDE POISONING
LEVELS OF CARBON MONOXIDE
LEVEL ASSESSMENT FINDING
5% to 10% Impaired visual acuity
11% to 20% Flushing
21% to 30% Nausea & impaired dexterity
31% to 40% Vomiting, dizziness, & syncope
41% to 50% Tachypnea & tachycardia
Ý 50% Coma & death
NURSING CARE
Remove victim from exposure
Administer O2
Assess for basic life support
V/S
Monitor carbon monoxide levels
HISTOPLASMOSIS
Caused by spores of Histoplasma capsulatum
Transmitted by inhalation of spores, which are commonly located in contaminated soil
Found in bird droppings
ASSESSMENT
Dyspnea
Chills
Fever
Chest pain
Pulmonary infiltrates on CXR
Elevated WBC
Splenomegaly & hepatomegaly
NURSING CARE
O2 as Rx
Monitor breath sounds
Antiemetics, antihistamines, antipyretics & corticosteroids as Rx
Fungicidal medication
Coughing & DBE
Ý Fowler’s
V/S
Monitor for nephrotoxicity
SARCOIDOSIS
Epitheloid cell tubercles in lung
Cause is unknown
ASSESSMENT
Night sweats
Fever
Weight loss
Cough
Skin nodules
Polyarthritis
NURSING CARE
Corticosteroids
Monitor temperature
Increase fluid intake
Provide frequent periods of rest
Encourage small, frequent meals
OCCUPATIONAL LUNG DISEASE : SILICOSIS
Known as ASBESTOSIS and COAL WORKER’S PNEUMONIA
- caused by the inhalation of inorganic dusts
- common in miners & sandblasters
- Tuberculosis (PTB) is a frequent complications
ASSESSMENT
Frequent respiratory infections
Bloody sputum
Cough
CXR: Nodular lesions of the lungs
NURSING CARE
Administer antitussive
Administer medication for TB as Rx
Eliminate the toxic substances
Administer O2 as Rx
Encourage coughing & DBE
ACID-BASE BALANCE
Ph – 7.35 – 7.45
ßph – acidosis ( Ý H ion conc.)
Ýph – alkalosis( ßH ion conc.)
BUFFER SYSTEM:
Bicarbonate Carbonic acid
HCO3 CO3
Strong base Weak acid
20 : 1
Normal ABG Values:
Ph : 7.35 – 7.45
PCO2 : 35 – 45 mm HG
HCO3 : 22-26 meq/L
PO2 : 85 – 100 mmHg
Base excess : (+2 or –2)
Respiratory Acidosis
1. Carbonic acid excess:
2. Increase retention of carbon dioxide
3. Pco2 is greater than 45 mm Hg
4. pH is below 7.45
Common causes:
1. Inadequate ventilation (dyspnea)
2. Respiratory obstruction (mechanical- tumor) (Functional – asthma)
3. Impaired gas exchange – (emphysema)
4. Neuromascular impairment – (spinal cord injury)
Signs of Resp. Acidosis
1. Dyspnea
2. Irritability
3. Disorientation
4. Tachycardia
5. Cyanosis
6. Coma
Compensatory mechanisms
1. The urinary system excretes increased hydrogen ions to compensate for the respiratory system’s to blow off CO2
2. The urinary system retains sodium to facilitate the body’s attempt to increase sodium bicarbonate
3. The rate and depth of respirations increase
4. With chronic hypoxia, decrease oxygen levels become the stimulant to breathe: normally, elevated CO2 level stimulate
breathing
Respiratory Alkalosis
1. Carbonic acid deficit
2. hyperventilation blows off excessive CO2
3. PCo2 is less than 35 mm Hg
4. pH is above 7.45
Common Causes
1. Hyperventilation related to anxiety/panic
2. Excessive mechanical ventilation
Signs and symptoms
1. Deep,rapid breathing
2. Lightheadedness
3. Tingling and numbness
4. Tinnitus
5. Loss of concentration
6. unconsciousness
Compensatory mechanism
• The urinary system may decrease the excretion of hydrogen ions to maintain the pH in the normal range.
Metabolic Acidosis
1. Base bicarbonate deficit
2. Excess acid other than carbonic acid (a respiratory acid) accumulates beyond the body’s ability to neutralize it;
3. Bicarbonate is below 22 mEq/L
4. ph is below 7.35
Common Causes
1. Cellular breakdown with increase ketones (Starvation, terminal CA, ketoacidosis, dieting)
2. Renal insufficiency (acute and chronic renal failure)
3. Lactic acid accumulation from anaerobic metabolism
Signs of Metabolic Acidosis
1. Weakness
2. Headache
3. Disorientation
4. Deep rapid breathing (kausmaul Respiration)
5. Fruity odor breath
6. Nausea and vomiting
7. coma
Compensatory mechanism
1. The respiratory system compensates by hyperventilation in an attempt to blow off CO2 and raise pH
2. The urinary system excretes hydrogen ions to remove excess hydrogen ions and sodium is retained to help increase
sodium bicarbonate
Metabolic Alkalosis
1. Base bicarbonate excess
2. Bicarbonate is above 26 mEq/L
3. pH is above 7.45
Common causes:
1. Loss of gastric juice (vomiting, nasogastric decompression, lavage)
2. Excessive ingestion of alkaline drugs sodiium bicarbonate (baking soda)
3. Potent diuretics may precipitate hypokalemia; in the presence of hypokalemia, the kidney conserve potassium and
excreate hydrogen ion
Sign of Metabolic Alkalosis
1. Muscle hypertonicity (tetany)
2. Tingling
3. Tremors
4. Shallow and slow respirations
5. Dizziness
6. Confusion
7. coma
Compensatory mechanisms of Metabolic alkalosis
1. The respiratory system compensate by decreasing the rate and depth of breathing to retain Co2 and decrease the pH
2. The urinary system excretes sodium bicarbonate