Anaemia Care Plan
Anaemia Care Plan
1. INTRODUCTION (SELF):-
As per my clinical posting I was posted in the Male Medicine ward in AGMC & GBP
Hospital. During my clinical posting I was found a patient who was suffering from
generalized weakness, drowsiness, breathing difficulty, mild fever, cough and doctor
diagnosed him as Chronic Anaemia. I had taken this diagnosis for my case presentation.
Age: 65 years
Gender: Male
Religion: Hindu
Occupation: Businessmen
C. R. No: 70540
Diagnosis: Anaemia
6. FAMILY HISTORY:
Types of Family: Nuclear
Family Chart:
2. Mrs. Namita 59 years Wife VIII passed House wife Married Healthy
Debnath. Female
4. Mrs. Kajal 27 years Daughter- XII passed House wife Married Healthy
Debnath. Female in- low
5. Mr. Tuhin 5 years Grandson Student Student Unmarried Healthy
Debnath. Male
Family Tree:
Key points
-Death Female
-Male patient
-Female
(Grandson, 5 yrs)
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7. PERSONAL HISTORY:
Nutritional Status: Undernourished
Habits: He is having the habit of bidi smoking and pan
Hygiene: Maintained properly
Allergens: He is not having any allergic reaction from anything.
Sleeping Pattern: He is having the habit of sleeping for 5-6 hours.
Bowel and Bladder Pattern: He passes stool and urine according to his body requirement.
Activities of Daily Living: Dull due to illness.
8. SOCIO-ECONOMIC HISTORY:
Head of the family: Mr. Narayan Ghosh.
Income: Approximately Rs. 20000/- per month
House Type: Pucca
Location: Urban
Water supply: Tap water
Electricity: Available
Ventilation: Good
Sanitation: Pucca
Drainage System: Pucca
9. PHYSICAL EXAMINATION:
Height: 145 cm.
Weight: 52 kg.
VITAL SIGNS:
GENERAL APPEARANCE:
Appearance: Dull
Health: Unhealthy
MENTAL STATUS:
POSTURE:
SKIN:
Colour: Brown
Texture: Dry
Temp.: 97.80 F
Pigmentation: Absent
EYES:
NECK:
CHEST:
Inspection:
Shape & Symmetry: Normal
Respiratory rate: 32 breaths/min.
Movements: Normal
Palpation:
There is no enlarged lymph nodes in the breast
Percussion:
Auscultation:
Breath Sound: Wheezing sound present.
Heart Sound: S1 and S2 sound heard.
ABDOMEN:
Inspection: There are no skin rashes, scar mark, lesions, and ascites. Abdominal
distension is present.
Auscultation: Bowel sound auscultated
Palpation: No organomegaly, splenomegaly found.
Percussion: There is no fluid accumulation.
EXTREMITIES:
Infection: No infection
Secretion: Absent
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Haemorrhoids: Absent.
12. INVESTIGATIONS:
Sl. Name of the Investigation Patient’s value Normal Value
No
.
01. Serum Urea 26 mg/dl 15-40 mg/dl
02. Serum Creatinine 0.9 mg/dl 0.6-1.5 mg/dl
03. SGPT (ALT) 20 IU/L 5-40 IU/L
04. SGOT (AST) 30 IU/L 5-40 IU/L
05. Serum Alkaline Phosphate(ALP) 127 IU/L 50-280 IU/L
06. Serum Na+ 130 mEq/L 135-145 mEq/L
07. Serum K+ 4.6 mEq/L 3.5-5.5 mEq/L
08. Blood glucose (Fasting) 108 mg/dl 90-110 mg/dl
09. Blood glucose (Post Prandial) 120 mg/dl 80-140mg/dl
10. Hb% 5.6 g/dl 13.0-17.0 g/dl
11. Uric acid 6.1 mg/dl 3.5- 7.2mg/dl
12. Chloride 94.8 mmol/L 98-107 mmol/L
13. Neutrophil 71% 40-60%
14. Lymphocyte 26% 18-45%
15. Monocyte 01% 2-8%
16. Eosinophil 02% 1-4%
17. Platelet count 1.1 lac 1.5-4 lac
18. HIV Non-reactive
19. HBsAg Non-reactive
20. HCV Non-reactive
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11. MEDICATION:
Sl. Dose, Nurses Responsibility
No. Drug name Action Route
Frequen
cy
1. Inj. Iron Iron sucrose injection is in a class of 20mg Monitor Bp during
Sucrose medications called iron replacement (Fe)/ml, infusion
products. It works by replenishing iron IV, Stat Assess for
stores so that the body can make more hypersensitivity
red blood cells. reactions and
anaphylaxis for at
least 30 min following
injection
2. Inj. EPO interacts directly with the EPO 4000 IU, Assess the general
Erythropoiet receptor on the red blood cell (RBC) Sc, Stat condition of the
in surface, triggering activation of several patient.
signal transduction pathways, resulting Monitor vital signs.
in the proliferation and terminal Don’t shake the vial
differentiation of erythroid precursor of Erythropoietin
cells and providing protection from Don’t administer
RBC precursor apoptosis. Erythropoietin in
conjunction with other
drug solution.
3 Tab. Nodosis tablet is used as an antacid that 500 mg, Check the diagnosis
Nodosis helps in neutralizing stomach acid Oral, and age of the patient.
during indigestion and heartburn. It TDS Monitor vitals signs
works by increasing pH in blood and Check the medication
urine, which corrects metabolic order
acidosis. Check the conscious
level of the patient
4 Tab. Shelcal It stimulates bone resorption, renal 500 mg, Monitor vital signs.
tubular resorption. The drug also Oral, OD Check any
decreases PTH levels and restores bone complication after
mineralization. Elemental Calcium is administering the
an antacid which works by neutralizing medicine.
the acid released in the stomach.
5 Tab. Rantac It reduce the secretion of gastric acid by 150 mg, Maintain the time.
reversible binding to histamine (H2) Oral, OD Check any side effect
receptors, which are found on gastric after administering the
parietal cells. This process leads to the medication.
inhibition of histamine binding to this
receptor, causing the reduction of
gastric acid secretion.
6. Tab. IFA An exogenous source of folate is Maintain vital signs.
required for nucleoprotein synthesis 120 mg, Assess general
(Iron Folic and the maintenance of normal Oral, condition of the
Acid) erythropoiesis. Folic acid, whether ODPC patient.
given by mouth or parenterally, Assess side effect
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1961 was from Teachers College, Columbia University, New York. She also did postdoctoral
study in research design, statistics and computers.
King had experience in nursing as an administrator, an educator and a practitioner. Her area
of clinical practice was adult medical-surgical nursing. She served as faculty member at St
John's Hospital School of Nursing, St Loyola University, Chicago, Illinois, and the
University of South Florida, Tampa. She was also the director of the School of Nursing at
Ohio State University, Imogene Columbus. She was an assistant chief of the Research Grants
Branch, Division of Nursing. Department of Health, Education and Welfare, in the mid 1960s
and on the Defence Advisory Committee on Women in the Services for the department of
Defense in the early 1970s.
Theory application
Mr. Priyotosh Debnath was admitted for the first time. He was anxious and very weak about
outcome of disease as well as adjusting in new environment. He was fully conscious, alert
and he can do care himself by minimal assistance. So, I applied King’s goal attainment theory
with the mutual understanding of patient and family members while caring him to improve
his health status by setting the goals with both the nurse and the patient’s mutual
understanding.
Perception
Judgement
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Nurse
Action Reaction
Interactio Transaction
n
Action Reaction
Perception
Client
Judgement
Feedback
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Patient’s
perception:-
Patient said “I am
Limited To Activity -Assess the activity level -Assessed the activity Patient’s
improve
suffering anaemia, Activity. the intoleranc of the patient. breathing
feeling weak and I level of the patient.
activity e related pattern are
can’t do work -Provide proper nutrition
properly”.
level. -Provided proper improved
to to the patient. gradually.
nutrition to the patient.
Nurse’s Perception:- weakness
Patient looks very -Administer IV fluid as
weak and he can’t as -Administered IV fluid
per doctor’s order.
work by own self. evidence as per doctor’s order.
-Provide medication as
Patient’s by patient -Provided medication
judgement:- per doctor’s order.
Patient understand the looks very as per doctor’s order.
need of taking care. dull. -Monitor vitals.
-Monitored vitals.
Nurse’s Judgement:- -Limit movement and
Based on the encourage the patient to
-Limited movement
assessment the
do ROM exercise and encourage the
diagnosis of this
problem is, “activity patient to do ROM
intolerance related to
weakness.” exercise.
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Subjective Imbalanced Improve the -Assess the nutrition level -Assess the nutrition level
Patient’s nutrition
data: nutrition of the patient. of the patient. pattern improve
nutrition
Patient said pattern. gradually.
that “I look less than -Provide proper nutrition -Provide proper nutrition to
slimmer that I the patient.
body to the patient.
was before
and my requirement -Instruct the patient to -Instruct the patient to avoid
weight also
related to avoid junk foods. junk foods.
reduce day by
day”. disease
-Check the weight of the -Check the weight of the
condition as patient.
patient.
Objective evidence by
data: -Administer IV fluid to -Administer IV fluid to the
reduce body
Patient looks patient.
the patient.
dull and weight.
weak. -Provide iron rich diet to -Provide iron rich diet to the
patient.
the patient.
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Medication:
Advised the patient relatives to give medicine at proper time without forgetting.
Encouraged the patient relatives to complete the full dose as ordered by the
physician/surgeon and to encourage him to promote activity level.
Follow up care:
Advised the patient relatives to bring the patient for regular check-up.
Advised the family members to provide physical, psychological support to the patient.
Encourage the patient relatives to provide a quiet and calm environment.
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16. Journal:
Abstract
Anemia affects a third of the world’s population and contributes to increased morbidity and
mortality, decreased work productivity, and impaired neurological development.
Understanding anemia’s varied and complex etiology is crucial for developing effective
interventions that address the context-specific causes of anemia and for monitoring anemia
control programs. We outline definitions and classifications of anemia, describe the
biological mechanisms through which anemia develops, and review the variety of conditions
that contribute to anemia development. We emphasize the risk factors most prevalent in low-
and middle-income countries, including nutritional deficiencies, infection/inflammation, and
genetic hemoglobin disorders. Recent work has furthered our understanding of anemia’s
complex etiology, including the proportion of anemia caused by iron deficiency (ID) and the
role of inflammation and infection. Accumulating evidence indicates that the proportion of
anemia due to ID differs by population group, geographical setting, infectious disease burden,
and the prevalence of other anemia causes. Further research is needed to explore the role of
additional nutritional deficiencies, the contribution of infectious and chronic disease, as well
as the importance of genetic hemoglobin disorders in certain populations.
17. CONCLUSION:-
As per my clinical posting I had posted in the Male Medicine ward and during my posting I
got a patient with Anaemia. I have given care as per the need of the patient and it will help
me to deal with the same kind of patient in future.
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17. REFERENCES:
Book:
Journal:
https://pubmed.ncbi.nlm.nih.gov/31008520/
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SUBMITTED TO SUBMITTED BY
MRS. GARGI MAITY BHOWMIK MS. JOYSREE BOSE.
ASSOCIATE PROFESSOR M.SC (N), 1ST SEM.
(DEPT. OF MEDICAL SURGICAL NURSING) ROLL NO:- 07.