Patient Name : Ms.
ANJALI DEVI Collected : 08/Mar/2024 09:16AM
Age/Gender : 51 Y 9 M 24 D /F Received : 08/Mar/2024 03:17PM
UHID/MR No : APJ1.0027164515 Reported : 08/Mar/2024 05:41PM
Visit ID : DPWBOPV23953 Status : Final Report
Ref Doctor : Dr.SELF Client Name : PUP 24X7_CREDIT
IP/OP NO : Center location : EPIP S.O,Bangalore
DEPARTMENT OF HAEMATOLOGY
APOLLO HEALTH CHECK - BASIC
Test Name Result Unit Bio. Ref. Interval Method
COMPLETE BLOOD COUNT (CBC) , WHOLE BLOOD EDTA
HAEMOGLOBIN 11.8 g/dL 12-15 Spectrophotometer
PCV 37.00 % 36-46 Electronic pulse &
Calculation
RBC COUNT 4.67 Million/cu.mm 3.8-4.8 Electrical Impedence
MCV 79.3 fL 83-101 Calculated
MCH 25.3 pg 27-32 Calculated
MCHC 31.9 g/dL 31.5-34.5 Calculated
R.D.W 16.6 % 11.6-14 Calculated
TOTAL LEUCOCYTE COUNT (TLC) 6,310 cells/cu.mm 4000-10000 Electrical Impedance
DIFFERENTIAL LEUCOCYTIC COUNT (DLC)
NEUTROPHILS 54.9 % 40-80 Electrical Impedance
LYMPHOCYTES 39.3 % 20-40 Electrical Impedance
EOSINOPHILS 3.2 % 1-6 Electrical Impedance
MONOCYTES 1.6 % 2-10 Electrical Impedance
BASOPHILS 1 % <1-2 Electrical Impedance
CORRECTED TLC 6,310 Cells/cu.mm Calculated
ABSOLUTE LEUCOCYTE COUNT
NEUTROPHILS 3464.19 Cells/cu.mm 2000-7000 Calculated
LYMPHOCYTES 2479.83 Cells/cu.mm 1000-3000 Calculated
EOSINOPHILS 201.92 Cells/cu.mm 20-500 Calculated
MONOCYTES 100.96 Cells/cu.mm 200-1000 Calculated
BASOPHILS 63.1 Cells/cu.mm 0-100 Calculated
Neutrophil lymphocyte ratio (NLR) 1.4 0.78- 3.53 Calculated
PLATELET COUNT 140000 cells/cu.mm 150000-410000 Electrical impedence
Result is rechecked. Kindly correlate clinically
Page 1 of 8
SIN No:HA06611498
Patient Name : Ms.ANJALI DEVI Collected : 08/Mar/2024 09:16AM
Age/Gender : 51 Y 9 M 24 D /F Received : 08/Mar/2024 03:42PM
UHID/MR No : APJ1.0027164515 Reported : 08/Mar/2024 05:44PM
Visit ID : DPWBOPV23953 Status : Final Report
Ref Doctor : Dr.SELF Client Name : PUP 24X7_CREDIT
IP/OP NO : Center location : EPIP S.O,Bangalore
DEPARTMENT OF BIOCHEMISTRY
APOLLO HEALTH CHECK - BASIC
Test Name Result Unit Bio. Ref. Interval Method
GLUCOSE, FASTING , NAF PLASMA 327 mg/dL 70-100 GOD - POD
Comment:
As per American Diabetes Guidelines, 2023
Fasting Glucose Values in mg/dL Interpretation
70-100 mg/dL Normal
100-125 mg/dL Prediabetes
≥126 mg/dL Diabetes
<70 mg/dL Hypoglycemia
Note:
1.The diagnosis of Diabetes requires a fasting plasma glucose of > or = 126 mg/dL and/or a random / 2 hr post glucose value of > or = 200 mg/dL on at least 2
occasions.
2. Very high glucose levels (>450 mg/dL in adults) may result in Diabetic Ketoacidosis & is considered critical.
Page 2 of 8
SIN No:BI18685108
Patient Name : Ms.ANJALI DEVI Collected : 08/Mar/2024 09:16AM
Age/Gender : 51 Y 9 M 24 D /F Received : 08/Mar/2024 03:42PM
UHID/MR No : APJ1.0027164515 Reported : 08/Mar/2024 05:44PM
Visit ID : DPWBOPV23953 Status : Final Report
Ref Doctor : Dr.SELF Client Name : PUP 24X7_CREDIT
IP/OP NO : Center location : EPIP S.O,Bangalore
DEPARTMENT OF BIOCHEMISTRY
APOLLO HEALTH CHECK - BASIC
Test Name Result Unit Bio. Ref. Interval Method
LIPID PROFILE , SERUM
TOTAL CHOLESTEROL 114 mg/dL <200 CHE/CHO/POD
TRIGLYCERIDES 102 mg/dL <150 Enzymatic
HDL CHOLESTEROL 53 mg/dL >40 CHE/CHO/POD
NON-HDL CHOLESTEROL 61 mg/dL <130 Calculated
LDL CHOLESTEROL 40.6 mg/dL <100 Calculated
VLDL CHOLESTEROL 20.4 mg/dL <30 Calculated
CHOL / HDL RATIO 2.15 0-4.97 Calculated
Comment:
Reference Interval as per National Cholesterol Education Program (NCEP) Adult Treatment Panel III Report.
Desirable Borderline High High Very High
TOTAL CHOLESTEROL < 200 200 - 239 ≥ 240
TRIGLYCERIDES <150 150 - 199 200 - 499 ≥ 500
Optimal < 100
LDL 130 - 159 160 - 189 ≥ 190
Near Optimal 100-129
HDL ≥ 60
Optimal <130;
NON-HDL CHOLESTEROL 160-189 190-219 >220
Above Optimal 130-159
1. Measurements in the same patient on different days can show physiological and analytical variations.
2. NCEP ATP III identifies non-HDL cholesterol as a secondary target of therapy in persons with high triglycerides.
3. Primary prevention algorithm now includes absolute risk estimation and lower LDL Cholesterol target levels to determine eligibility of drug therapy.
4. Low HDL levels are associated with Coronary Heart Disease due to insufficient HDL being available to participate in reverse cholesterol transport, the process by
which cholesterol is eliminated from peripheral tissues.
5. As per NCEP guidelines, all adults above the age of 20 years should be screened for lipid status. Selective screening of children above the age of 2 years with a family
history of premature cardiovascular disease or those with at least one parent with high total cholesterol is recommended.
6. VLDL, LDL Cholesterol Non HDL Cholesterol, CHOL/HDL RATIO, LDL/HDL RATIO are calculated parameters when Triglycerides are below 400 mg/dL. When
Triglycerides are more than 400 mg/dL LDL cholesterol is a direct measurement.
Page 3 of 8
SIN No:BI18685109
Patient Name : Ms.ANJALI DEVI Collected : 08/Mar/2024 09:16AM
Age/Gender : 51 Y 9 M 24 D /F Received : 08/Mar/2024 03:42PM
UHID/MR No : APJ1.0027164515 Reported : 08/Mar/2024 05:44PM
Visit ID : DPWBOPV23953 Status : Final Report
Ref Doctor : Dr.SELF Client Name : PUP 24X7_CREDIT
IP/OP NO : Center location : EPIP S.O,Bangalore
DEPARTMENT OF BIOCHEMISTRY
APOLLO HEALTH CHECK - BASIC
Test Name Result Unit Bio. Ref. Interval Method
LIVER FUNCTION TEST (LFT) , SERUM
BILIRUBIN, TOTAL 0.30 mg/dL 0.20-1.20 DIAZO METHOD
BILIRUBIN CONJUGATED (DIRECT) 0.10 mg/dL 0.0-0.3 Calculated
BILIRUBIN (INDIRECT) 0.20 mg/dL 0.0-1.1 Dual Wavelength
ALANINE AMINOTRANSFERASE 28 U/L 9-52 UV with P-5-P
(ALT/SGPT)
ASPARTATE AMINOTRANSFERASE 31.0 U/L 14-36 UV with P-5-P
(AST/SGOT)
ALKALINE PHOSPHATASE 136.00 U/L 38-126 p-nitrophenyl
phosphate
PROTEIN, TOTAL 6.90 g/dL 6.3-8.2 Biuret
ALBUMIN 4.10 g/dL 3.5 - 5 Bromocresol Green
GLOBULIN 2.80 g/dL 2.0-3.5 Calculated
A/G RATIO 1.46 0.9-2.0 Calculated
Comment:
LFT results reflect different aspects of the health of the liver, i.e., hepatocyte integrity (AST & ALT), synthesis and secretion of bile (Bilirubin, ALP), cholestasis (ALP,
GGT), protein synthesis (Albumin)
Common patterns seen:
1. Hepatocellular Injury:
• AST – Elevated levels can be seen. However, it is not specific to liver and can be raised in cardiac and skeletal injuries.
• ALT – Elevated levels indicate hepatocellular damage. It is considered to be most specific lab test for hepatocellular injury. Values also correlate well with increasing
BMI .• Disproportionate increase in AST, ALT compared with ALP. • Bilirubin may be elevated.
• AST: ALT (ratio) – In case of hepatocellular injury AST: ALT > 1In Alcoholic Liver Disease AST: ALT usually >2. This ratio is also seen
to be increased in NAFLD, Wilsons’s diseases, Cirrhosis, but the increase is usually not >2.
2. Cholestatic Pattern:
• ALP – Disproportionate increase in ALP compared with AST, ALT.
• Bilirubin may be elevated.• ALP elevation also seen in pregnancy, impacted by age and sex.
• To establish the hepatic origin correlation with GGT helps. If GGT elevated indicates hepatic cause of increased ALP.
3. Synthetic function impairment: • Albumin- Liver disease reduces albumin levels.• Correlation with PT (Prothrombin Time) helps.
Page 4 of 8
SIN No:BI18685109
Patient Name : Ms.ANJALI DEVI Collected : 08/Mar/2024 09:16AM
Age/Gender : 51 Y 9 M 24 D /F Received : 08/Mar/2024 03:42PM
UHID/MR No : APJ1.0027164515 Reported : 08/Mar/2024 05:44PM
Visit ID : DPWBOPV23953 Status : Final Report
Ref Doctor : Dr.SELF Client Name : PUP 24X7_CREDIT
IP/OP NO : Center location : EPIP S.O,Bangalore
DEPARTMENT OF BIOCHEMISTRY
APOLLO HEALTH CHECK - BASIC
Test Name Result Unit Bio. Ref. Interval Method
RENAL PROFILE/KIDNEY FUNCTION TEST (RFT/KFT) , SERUM
CREATININE 0.40 mg/dL 0.5-1.04 Creatinine
amidohydrolase
UREA 12.84 mg/dL 15-36 Urease
BLOOD UREA NITROGEN 6.0 mg/dL 8.0 - 23.0 Calculated
URIC ACID 2.40 mg/dL 2.5-6.2 Uricase
CALCIUM 8.80 mg/dL 8.4 - 10.2 Arsenazo-III
PHOSPHORUS, INORGANIC 3.30 mg/dL 2.5-4.5 PMA Phenol
SODIUM 145 mmol/L 135-145 Direct ISE
POTASSIUM 3.8 mmol/L 3.5-5.1 Direct ISE
CHLORIDE 110 mmol/L 98 - 107 Direct ISE
PROTEIN, TOTAL 6.90 g/dL 6.3-8.2 Biuret
ALBUMIN 4.10 g/dL 3.5 - 5 Bromocresol Green
GLOBULIN 2.80 g/dL 2.0-3.5 Calculated
A/G RATIO 1.46 0.9-2.0 Calculated
Page 5 of 8
SIN No:BI18685109
Patient Name : Ms.ANJALI DEVI Collected : 08/Mar/2024 09:16AM
Age/Gender : 51 Y 9 M 24 D /F Received : 08/Mar/2024 03:42PM
UHID/MR No : APJ1.0027164515 Reported : 08/Mar/2024 06:24PM
Visit ID : DPWBOPV23953 Status : Final Report
Ref Doctor : Dr.SELF Client Name : PUP 24X7_CREDIT
IP/OP NO : Center location : EPIP S.O,Bangalore
DEPARTMENT OF IMMUNOLOGY
APOLLO HEALTH CHECK - BASIC
Test Name Result Unit Bio. Ref. Interval Method
THYROID PROFILE TOTAL (T3, T4, TSH) , SERUM
TRI-IODOTHYRONINE (T3, TOTAL) 0.86 ng/mL 0.7-2.04 CLIA
THYROXINE (T4, TOTAL) 9.48 µg/dL 5.48-14.28 CLIA
THYROID STIMULATING HORMONE 2.573 µIU/mL 0.34-5.60 CLIA
(TSH)
Comment:
Bio Ref Range for TSH in uIU/ml (As per American
For pregnant females
Thyroid Association)
First trimester 0.1 - 2.5
Second trimester 0.2 – 3.0
Third trimester 0.3 – 3.0
1. TSH is a glycoprotein hormone secreted by the anterior pituitary. TSH activates production of T3 (Triiodothyronine) and its prohormone T4 (Thyroxine).
Increased blood level of T3 and T4 inhibit production of TSH.
2. TSH is elevated in primary hypothyroidism and will be low in primary hyperthyroidism. Elevated or low TSH in the context of normal free thyroxine is often
referred to as sub-clinical hypo- or hyperthyroidism respectively.
3. Both T4 & T3 provides limited clinical information as both are highly bound to proteins in circulation and reflects mostly inactive hormone. Only a very small
fraction of circulating hormone is free and biologically active.
4. Significant variations in TSH can occur with circadian rhythm, hormonal status, stress, sleep deprivation, medication & circulating antibodies.
TSH T3 T4 FT4 Conditions
High Low Low Low Primary Hypothyroidism, Post Thyroidectomy, Chronic Autoimmune Thyroiditis
Subclinical Hypothyroidism, Autoimmune Thyroiditis, Insufficient Hormone Replacement
High N N N
Therapy.
N/Low Low Low Low Secondary and Tertiary Hypothyroidism
Low High High High Primary Hyperthyroidism, Goitre, Thyroiditis, Drug effects, Early Pregnancy
Low N N N Subclinical Hyperthyroidism
Low Low Low Low Central Hypothyroidism, Treatment with Hyperthyroidism
Low N High High Thyroiditis, Interfering Antibodies
N/Low High N N T3 Thyrotoxicosis, Non thyroidal causes
High High High High Pituitary Adenoma; TSHoma/Thyrotropinoma
Page 6 of 8
SIN No:IM07108217
Patient Name : Ms.ANJALI DEVI Collected : 08/Mar/2024 09:16AM
Age/Gender : 51 Y 9 M 24 D /F Received : 08/Mar/2024 03:42PM
UHID/MR No : APJ1.0027164515 Reported : 08/Mar/2024 10:42PM
Visit ID : DPWBOPV23953 Status : Final Report
Ref Doctor : Dr.SELF Client Name : PUP 24X7_CREDIT
IP/OP NO : Center location : EPIP S.O,Bangalore
DEPARTMENT OF IMMUNOLOGY
VITAMIN-D FEMALE
Test Name Result Unit Bio. Ref. Interval Method
VITAMIN D (25 - OH VITAMIN D) , 8.87 ng/mL CLIA
SERUM
Comment:
BIOLOGICAL REFERENCE RANGES
VITAMIN D STATUS VITAMIN D 25 HYDROXY (ng/mL)
DEFICIENCY <10
INSUFFICIENCY 10 – 30
SUFFICIENCY 30 – 100
TOXICITY >100
The biological function of Vitamin D is to maintain normal levels of calcium and phosphorus absorption. 25-Hydroxy vitamin D is the storage form of vitamin D.
Vitamin D assists in maintaining bone health by facilitating calcium absorption. Vitamin D deficiency can also cause osteomalacia, which frequently affects elderly
patients.
Vitamin D Total levels are composed of two components namely 25-Hydroxy Vitamin D2 and 25-Hydroxy Vitamin D3 both of which are converted into active
forms. Vitamin D2 level corresponds with the exogenous dietary intake of Vitamin D rich foods as well as supplements. Vitamin D3 level corresponds with
endogenous production as well as exogenous diet and supplements.
Vitamin D from sunshine on the skin or from dietary intake is converted predominantly by the liver into 25-hydroxy vitamin D, which has a long half-life and is
stored in the adipose tissue. The metabolically active form of vitamin D, 1,25-di-hydroxy vitamin D, which has a short life, is then synthesized in the kidney as
needed from circulating 25-hydroxy vitamin D. The reference interval of greater than 30 ng/mL is a target value established by the Endocrine Society.
Decreased Levels:
Inadequate exposure to sunlight.
Dietary deficiency.
Vitamin D malabsorption.
Severe Hepatocellular disease.
Drugs like Anticonvulsants.
Nephrotic syndrome.
Increased levels:
Vitamin D intoxication.
Page 7 of 8
SIN No:IM07108217
Patient Name : Ms.ANJALI DEVI Collected : 08/Mar/2024 09:16AM
Age/Gender : 51 Y 9 M 24 D /F Received : 08/Mar/2024 07:08PM
UHID/MR No : APJ1.0027164515 Reported : 08/Mar/2024 07:36PM
Visit ID : DPWBOPV23953 Status : Final Report
Ref Doctor : Dr.SELF Client Name : PUP 24X7_CREDIT
IP/OP NO : Center location : EPIP S.O,Bangalore
DEPARTMENT OF CLINICAL PATHOLOGY
APOLLO HEALTH CHECK - BASIC
Test Name Result Unit Bio. Ref. Interval Method
COMPLETE URINE EXAMINATION (CUE) , URINE
PHYSICAL EXAMINATION
COLOUR PALE YELLOW PALE YELLOW Visual
TRANSPARENCY SIGHTLY TURBID CLEAR Visual
pH 6.0 5-7.5 Bromothymol Blue
SP. GRAVITY 1.015 1.002-1.030 Dipstick
BIOCHEMICAL EXAMINATION
URINE PROTEIN POSITIVE (TRACE) NEGATIVE PROTEIN ERROR OF
INDICATOR
GLUCOSE POSITIVE (++) NEGATIVE GOD-POD
URINE BILIRUBIN NEGATIVE NEGATIVE AZO COUPLING
URINE KETONES (RANDOM) NEGATIVE NEGATIVE NITROPRUSSIDE
UROBILINOGEN NORMAL NORMAL EHRLICH
BLOOD NEGATIVE NEGATIVE Dipstick
NITRITE POSITIVE NEGATIVE Dipstick
LEUCOCYTE ESTERASE POSITIVE NEGATIVE PYRROLE
HYDROLYSIS
CENTRIFUGED SEDIMENT WET MOUNT AND MICROSCOPY
PUS CELLS 10-12 /hpf 0-5 Microscopy
EPITHELIAL CELLS 15-16 /hpf <10 MICROSCOPY
RBC NIL /hpf 0-2 MICROSCOPY
CASTS NIL 0-2 Hyaline Cast MICROSCOPY
CRYSTALS ABSENT ABSENT MICROSCOPY
OTHERS BACTERIA SEEN MICROSCOPY
*** End Of Report ***
Page 8 of 8
SIN No:C02660862
Patient Name : Ms.ANJALI DEVI Collected : 08/Mar/2024 09:16AM
Age/Gender : 51 Y 9 M 24 D /F Received : 08/Mar/2024 07:08PM
UHID/MR No : APJ1.0027164515 Reported : 08/Mar/2024 07:36PM
Visit ID : DPWBOPV23953 Status : Final Report
Ref Doctor : Dr.SELF Client Name : PUP 24X7_CREDIT
IP/OP NO : Center location : EPIP S.O,Bangalore
TERMS AND CONDITIONS GOVERNING THIS REPORT
1. Reported results are for information and interpretation of the referring doctor or such other medical professionals,
who understandreporting units, reference ranges and limitation of technologies.Laboratories not be responsible for any
interpretation whatsoever.
2. It is presumed that the tests performed are, on the specimen / sample being to the patient named or identified and the
verifications of parrticulars have been confirmed by the patient or his / her representative at the point of generation of said specimen.
3. The reported results are restricted to the given specimen only. Results may vary from lab to lab and from time to time for the same
parameter for the same patient (within subject biological variation).
4. The patient details along with their results in certain cases like notifiable diseases and as per local regulatory requirements will be
communicated to the assigned regulatory bodies.
5. The patient samples can be used as part of internal quality control, test verification, data analysis purposes within the testing scope of
the laboratory.
6. This report is not valid for medico legal purposes. It is performed to facilitate medical diagnosis only.
SIN No:C02660862
