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Paraplegia

This document discusses paraplegia, which is paralysis or weakness of both lower limbs. It can be spastic paraplegia due to damage to the upper motor neurons in the spinal cord, or flaccid paraplegia due to damage to the lower motor neurons. Spastic paraplegia can be with or without a sensory level. Causes of spastic paraplegia include spinal cord compression, inflammation, vascular issues, and tumors. Clinical features include motor deficits below the lesion level, sensory changes depending on lesion location, and sphincter issues. Investigations include imaging of the spine and neurological testing. Management involves nursing care, physiotherapy, treating underlying causes, and preventing complications.

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100% found this document useful (2 votes)
1K views56 pages

Paraplegia

This document discusses paraplegia, which is paralysis or weakness of both lower limbs. It can be spastic paraplegia due to damage to the upper motor neurons in the spinal cord, or flaccid paraplegia due to damage to the lower motor neurons. Spastic paraplegia can be with or without a sensory level. Causes of spastic paraplegia include spinal cord compression, inflammation, vascular issues, and tumors. Clinical features include motor deficits below the lesion level, sensory changes depending on lesion location, and sphincter issues. Investigations include imaging of the spine and neurological testing. Management involves nursing care, physiotherapy, treating underlying causes, and preventing complications.

Uploaded by

rashid38
Copyright
© Attribution Non-Commercial (BY-NC)
We take content rights seriously. If you suspect this is your content, claim it here.
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Download as PPT, PDF, TXT or read online on Scribd
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PARAPLEGIA

Presented By \
Dr. KAMAL OSMAN
MIRGANI
Is paralysis or weakness **
. ( paraparesis ) of both lower limbs

It is either :Spastic paraplegia due to **


pyramidal lesion (U.M.N.L )

Flaccid paraplegia ( L . M . N. L ) **
Spastic Paraplegia

: Definition 

Paralysis or weakness of both lower limbs


due to bilateral pyramidal tract lesion , most
commonly in the spinal card (spinal
paraplegia ) and less commonly in the brain
stem or cerebral parasagittal region ( cerebral
) paraplegia
-: Spastic Paraplegia

it either ***
. with sensory level
. with out sensory level
Spastic Paraplegia with sensory
level
: causes
: Cord compression - 1
: A-vertebral
fracture or fracture dislocation of vertebra*
Disc prolapse and spandylosis*
Pott’s disease of the spine*
Neoplasms : A 1ry – osteosarcoma*
haemangioma
MRI : acute cervical c5\6 soft disc prolapse
B 2ndary metastatic deposite from eg
breast - lung- stomach – prostate –
.kidney
: A-Extramedullary causes
Extradural eg , leukaemic deposits **
** Dural eg , menigioma
pachymeningitis
** Intradural eg , neurofibroma
: B- Intramedullary causes
Syringiomyelia – Gloom – Ependymoma
of cord
: Inflammatory 2
transverse myelitis
. myelomeningitis
: Vascular 3
. anterior spinal artery occlusion
Spinal paraplegia with out sensory
level

. Subcombined degeneration of the cord


. Motor neuron disease
. Hereditary spastic paraplegia
. Friedreich ataxia
NEXT picture shows
narrowing of intervertebral
disc space
NEXT picture shows
extramedullary , intradural
neurofibroma
NEXT picture shows
intramrdullary cervical cord
glioma
.Multiple sclerosis

.Disseminated encephalomyelitis

. Syphilis
Causes in parasagittal region ( area of
.) cortical presentation of L . L
Depressed fracture of the vault of
. skull causing subdural haematoma
. Superior sagittal sinus Thrombosis
. Parasagittal meningioma
. e. mid brain stem tumor
REMEMBER The
Differences between
EXTRA- & SUB- dural
haematomas
In the NEXT PICTUREs
WHAT is your diagnosis?
That was right frontoparietal
extradural haematomadue to a
squamous temporal fracture
not visible on CT
THAT was axial CT showing a
rghit-sided acute sub dural
haematoma with midline shift
Removal of acute
extradural haematoma by
Craniotomy
CLINICAL FEATURES
At the level of the lesion
there is localized tenderness or pain **
or deformity or swelling if the
. causes of vertebral causes
Radicular pain only seen in **
extramedulluy causes
Below the level of the lesion ( cord
) Manifestations
Motor manifestation .1
,A .If the causes acute ( inflamtion , vascular
) traumatic
: The paraplegia passes into 2 stages
: Stage of flaccidity ] 1 [
due to neuronal Shock complete *
paralysis of L . L with absence of reflexes
lasts from 2 – 6weeks *

:Stage of spasticity ]2[
due to recovery form neuronal
shock and full picture of
, UMNL will be established eg
hypertonia , Babaniski sign +ve and
.may be clonus
B – If the cause is gradual . (eg ,
:neoplastic )
There will be progressive
weakness of the lower limb, with
. hypertonia and hypereflexia
The Anatomy Of The Spinal Cord
Sensory manifestation – 2
a . if the cause of the lesion is
extramedullary
encroachment on the ascending tract , at
the site of the lesion result in sensory
level below which all types of sensation
. are diminished
There is early loss of sensation in
the saddle area ( S 3 , 4 , 5 ) as the
sacral fibres lie in the outer most part
.of the spinothalamic tract in the cord
B. if the cause of the lesion is
: intramedullay
there will be hyposthetic area with **
normal sensation above and below the
lesion ( Jacket sensory loss )
the sensory loss is of disassociated **
nature ie pain and temperature
sensations are lost but touch and
. deep sensations are preserved **
This due to the interruption of
crossing fibers carrying pain and
temperature by midline lesion while
touch and deep sensation fibers
ascend in the posterior
column with decussating . The
sensation over the saddle area are
preserved as sacral fibers , lie far
. from the midline
Sphincter manifestation – 3

a . in the acute lesions


retention of urine in the shock
stage , followed by precipitancy of
. micturation
a . in the acute lesions
retention of urine in the shock
stage , followed by precipitancy of
. micturation
: b. In gradual lesion
Precipitancy of micturation which may
terminated in autonomic bladder when
complete transection of cord
. occurs
The changes started later in extameduallay
lesions as the pyramidal fibers controlling
. the bladder center lie medially in the cord
INVESTIGATIONS
. Complete haemogram + E S R 1
. X –ray chest 2
C. S. F analysis 3
. C. T . scan 4
. X – ray spines 5
.M R I of vertebral column 6
. Vitamin B12 assay 7
MANAGEMENT OF
PARAPLEGIA
: A General : nursing care

Frequent turning of pt to prevent bed 1


. sores and Hypostatic state
Care of skin frequent washing with 2
.alcohol and talk power
. Care of the bladder eg catheterization 3
:Physiotherapy 4
Massage to increase blood supply to **
. paralysed muscles
Positioning: the paralysed limbs **
.should be put opposite to hypertonia
Passive excercise to gurd against **
. fibrosis and stiffness of joints
. Good adequate nutrition 5
pressure sore locations

                                                       

B TREATMENT :
of complications
.
C Specific treatment of the 
cause
. Anituberculous for pott’s disease .1
Deep–radiation for intramedullary .2
. tumors
Surgical excision for extramedllary .3
. tumours
,,,,,,,,,,,,,,,,,,,,,,,.With My Best Wishes

Dr \ KAMAL
OSMAN
MIRGANI

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