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Immediate Life Support - New

This document provides information on immediate life support (ILS) training. It discusses the ABCDE approach to assessing and treating deteriorating patients. The causes of airway, breathing, and circulation problems are outlined. The importance of prevention through monitoring, recognition of deterioration, and effective communication is emphasized. Cardiac arrest management focuses on high-quality chest compressions and the full ABCDE assessment and treatment protocol.

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0% found this document useful (1 vote)
1K views67 pages

Immediate Life Support - New

This document provides information on immediate life support (ILS) training. It discusses the ABCDE approach to assessing and treating deteriorating patients. The causes of airway, breathing, and circulation problems are outlined. The importance of prevention through monitoring, recognition of deterioration, and effective communication is emphasized. Cardiac arrest management focuses on high-quality chest compressions and the full ABCDE assessment and treatment protocol.

Uploaded by

Talha
Copyright
© © All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as PPT, PDF, TXT or read online on Scribd
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IMMEDIATE LIFE

SUPPORT
LEARNING OBJECTIVES

 What is ILS?

 Causes & Prevention of Cardiac arrest

 ABCDE approach

 Cardiac arrest

 Cardiac Arrest moulages


WHAT IS ILS?

 Provides knowledge and skills needed to treat


adult patients in cardio-respiratory arrest for
the short time before the arrival of a
resuscitation team or more experienced
assistance

 Teaches the recognition and treatment of the


deteriorating patient using the ABCDE
approach.
ABCDE APPROACH

 A – Airway

 B – Breathing

 C – Circulation

 D – Disability

 E - Exposure
CHAIN OF SURVIVAL
CAUSES AND PREVENTION OF
CARDIORESPIRATORY ARREST
WHY IS PREVENTION OF CARDIO-
RESPIRATORY ARREST IMPORTANT?

 Once cardiac arrest occurs, fewer than 20% of


patient having an in-hospital cardiac arrest will
survive to go home.

 Most in-hospital cardiac arrests are not sudden


or unpredictable events. In approximately 80%
of cases clinical signs deteriorate over the few
hours before arrest.
CHAIN OF PREVENTION
EDUCATION
 How to observe patients

 Recognising signs of deterioration

 Using the ABCDE approach

 Simple skills to stabilise the patient pending the


arrival of more experienced help
MONITORING

 Patient assessment

 Measurement and recording of vital signs


RECOGNITION & CALL FOR HELP

 To identify patients in need of additional monitoring or


intervention (e.g Early Warning scoring system)

 Protocols for summoning a response to a deteriorating


patient (clear deterioration prompts early help)

 Using a structured communication tool such as SBAR or


RSVP to call for help
EARLY WARNING SCORING SYSTEM

Score 3 2 1 0 1 2 3

<45 30 15% Normal for 15% >4


Systolic BP 30%
% % down patient up 5%

<4 101- 111- >1


Heart rate (BPM) — 41-50 51-100
0 110 129 30

Respiratory rate 15- >3


— <9 — 9-14 21-29
(RPM) 20 0

<3
Temperature (°C) — — 35.0-38.4 — >38.5 —
5

AVPU — — — A V P U
ESCALATION PROTOCOL BASED ON EWS
EWS Minimal Observation Escalation
frequency
Recorder’s action Doctor’s action

3-5 4 hourly Inform nurse in charge

6 4 hourly Inform doctor Doctor to see within 1 hour

7-8 1 hourly Inform doctor Doctor to see within 30


minutes and discuss with
senior doctor and / or
Consider continuous
monitoring outreach team

>9 30 minutes Inform doctor Doctor to see within 15


minutes and discuss with
senior doctor and ICU team
Start continuous monitoring
CALL FOR HELP

SBAR RSVP

SITUATION REASON

BACKGROUND STORY

ASSESSMENT VITAL SIGNS

RECOMMENDATION PLAN
RESPONSE

 Assured

 Specified speed

 Appropriate personnel (e.g. ICU team)


EXAMPLE
CAUSES OF DETERIORATION AND CARDIO-
RESPIRATORY ARREST

AIRWAY

BREATHING

CIRCULATION
AIRWAY OBSTRUCTION

Causes
 CNS depression (loss of airway patency and
protective reflexes)
 Blood, vomitus, foreign body

 Trauma to face or throat

 Epiglottitis

 Pharyngeal swelling (e.g infection, oedema)

 Laryngospasm, bronchospasm

 Bronchial secretions

 Blocked teacheostomy or laryngectomy


AIRWAY OBSTRUCTION (contd…)

Recognition

 Conscious patient Difficulty breathing, choking,


distressed

 Partial obstruction  NOISY

 Complete obstruction  SILENT, no air


movement at mouth

 Accessory muscle usage, see-saw respiration


AIRWAY OBSTRUCTION (contd…)

Treatment

 Treat the cause (e.g suction)

 Protect airway
 airway opening manoeuvres (head tilt / chin lift
or jaw thrust)
 insertion of adjuncts (e.g. oropharyngeal)
 intubation

 Oxygen
BREATHING PROBLEMS

Causes

 Acute or Chronic

 Lung disorder  infection, COPD, hemo /


pneumothorax, embolism

 Respiratory drive  CNS depression

 Respiratory effort  Spinal cord damage, diseases


(myasthenia gravis, GBS), pain
BREATHING PROBLEMS (contd…)
Recognition

 Conscious patient  shortness of breath

 Irritability, confusion, lethargy, reduced consciousness

 Cyanosis – LATE sign

 Indicators:
 R/R
 Pulse Oximetry
 ABG’s
BREATHING PROBLEMS (contd…)

Treatment

 Oxygen
 High flow to start
 Aim for 94-98% saturation on stability
 COPD – 88 – 92% saturation on stability

 Treat the cause

 Non-invasive ventilation

 Controlled ventilation (requiring intubation)


CIRCULATION PROBLEMS

Causes

 Primary Heart Problems  ACS, valvular, etc

 Secondary Heart Problems


 Respiratory arrest causing lack of oxygenation
 Anemia

 Septic shock

 Hypothermia
CIRCULATION PROBLEMS (contd…)

Recognition
 Chest pain
 Shortness of breath
 Syncope (fainting)
 Tachycardia / bradycardia
 Tachypnea
 Hypotension
 Poor peripheral perfusion
 Altered mental status
 Oliguria
CIRCULATION PROBLEMS (contd…)
Treatment

 ECG

 Treat the cause  I/V fluids for hypotension

 For ACS:
 Sit patient up
 Aspirin 300mg
 Nitroglycerine (unless hypotensive)
 Oxygen
 Morphine for relief of pain
 If STEMI  decide for thrombolysis / PCI
ANY QUESTIONS?
SUMMARY

 Causes of deterioration in patients

 Importance of vital observations

 Good and early communication

 Rapid initiation of treatment


ABCDE APPROACH
A - AIRWAY
Assess
 see-saw respiration, accessory muscle usage
 cyanosis
 noisy / silent or no breath sounds
 depressed consciousness

Treat
 Airway opening maneuvres (head tilt, chin lift / jaw
thrust)
 Suction
 Insertion of airway adjunct / intubation (expert only)
 Oxygen
B - BREATHING

Look, Listen, Feel


 R/R
 Depth, chest expansion

 Chest deformity

 sweating, central cyanosis, accessory muscle


usage, abdominal breathing
 Added sounds

 Percussion + Auscultation

 Tracheal position

 Chest wall palpation


B – BREATHING (contd…)

Treat

 Oxygen (for COPD aim for 88-92%)


 Needle thoracosotomy, chest drain insertion
C - CIRCULATION
Assess

 Colour and feel of hands


 Capillary refill time
 Pulse rate
 Character of peripheral and central pulses
 Blood pressure
 Auscultate the heart
 Conscious level
 Urine output (if catheterised)
 Ongoing bleeding, collections in drains
C – CIRCULATION (contd…)
Treat

 Insert 14 or 16 G I/V cannula (one or more)

 Take blood samples for investigations and group and


save

 Rapid fluid challenge of N/saline or Hartmann’s:


 If normotensive - 500 ml

 If hypotensive – 1 liter

 If known cardiac failure - smaller volumes (250ml) with


closer monitoring
C – CIRCULATION (contd…)

Treat

 Control bleeding

 Reassess and repeat fluid challenge if no


improvement

 Reassess and ask for help if no improvement


(inotropes or vasopressors maybe needed)

 Get ECG and follow ACS protocol if appropriate


D - DISABILITY
Assess

 Review ABCs

 Consciousness level
 A – Alert
 V – responds to Vocal stimulus
 P – responds to Painful stimulus
 U – Unresponsive
 Or GCS

 Pupils

 Blood GLUCOSE

 Drug induced causes

 Lateral position if airway not protected


E - EXPOSURE

 Full exposure of body


 Respect patient’s dignity

 Minimise heat loss


ALWAYS

 Remain systematic in approach


 First initiate treatment for identified problem,
then move to next step
 Reassess and review your interventions
starting from A going to E
 Don’t Ever Forget Glucose
 Obtain history from patient, relative, staff,
notes
 Document assessment, intervention and
responses
ANY QUESTIONS?
SUMMARY

 Systematic structured ABCDE approach

 Importance of treatment with identification

 DEF – Glucose

 Continuous re-assessments
CARDIAC ARREST
CHEST COMPRESSIONS
 Correct hand position – middle of the lower half of
the sternum

 High quality compressions:


 Depth of 5-6 cm
 Rate of 100-120 compressions per minute
 Allow the chest to recoil completely after each
compression
 Minimise interruptions. Plan when and why to pause

 Do not feel for pulse to assess compression


effectiveness

 If intubated, continue compressions uninterrupted


ADVANCED LIFE SUPPORT
 Standardised approach to patient with cardio-
respiratory arrest.

 Team members can predict and prepare for the next


stage in patient’s treatment

 Most important interventions that improve survival


after cardiac arrest are:
 Early, uninterrupted high quality chest compressions
 Early defibrillation for VF / VT

 Drugs and advanced airways are of secondary


importance
VENTRICULAR TACHYARDIA
VENTRICULAR FIBRILLATION
FINE VENTRICULAR FIBRILLATION
ASYSTOLE
PULSELESS ELECTRICAL ACTIVITY
ADVANCED LIFE SUPPORT - SHOCKABLE

 Interval between stopping compressions and


delivering a shock must not be more than 5
seconds ideally

 Resume chest compressions immediately after


a shock without checking the rhythm or a pulse

 Give adrenaline 1mg (1:10000) I/V after 3 rd


shock. Repeat if arrest persists every alternate
cycle.
ADVANCED LIFE SUPPORT – SHOCKABLE (contd…)

 Give amiodarone 300mg I/V after 3 rd shock.


Give further 150mg I/V if VF / VT persists.

 If amiodarone is unavailable, consider 100mg


Lidocaine. Give an additional 50mg if
necessary

 If rhythm is organised – check for pulse.


ADVANCED LIFE SUPPORT – NON-SHOCKABLE

 Pulseless Electrical Activity: organised cardiac


‘electrical activity’ in the absence of palpable
pulses.

 Give adrenaline 1mg (1:10000) I/V as soon as


intravascular access is achieved. Repeat if
arrest persists every alternate cycle.
DURING CPR

 Give good quality chest compressions

 Recognise and treat reversible causes

 Obtain a secure airway

 Obtain a vascular access


AIRWAY AND VENTILATION

 Use a bag mask / supraglottic airway device

 Ventilate the lungs at 10 breaths / minute

 Tracheal intubation should only be attempted


by trained healthcare provider

 Once intubated  continue chest compressions


at a rate of 100-120 without pausing ventilation
VASCULAR ACCESS

 Peripheral venous cannulation is quicker,


easier and safer.

 Give flush of 20 ml of fluid post drug


administration. Elevate the extremity for 10-20
seconds.

 If I/V access cannot be established, consider


intraosseous access.
REVERSIBLE CAUSES

4 H’S

 Hypoxia

 Hypovolaemia

 Hypoglycemia / hypo- or hyper- kalemia /


hypocalcemia / acidaemia / metabolic disorders

 Hypothermia
REVERSIBLE CAUSES (Contd…)

4 T’s

 Tension pneumothorax

 Tamponade

 Toxins

 Thrombosis (pulmonary or coronary thrombosis)


SIGNS OF LIFE

 Respiratory effort

 Movement

 Check for organised rhythm

 Check for pulse

 If no pulse – continue CPR


CONTINUE RESUSCITATION UNTIL

 Qualified / senior help arrives and takes over

 The patient starts to show signs of regaining


consciousness, such as coughing, opening his
eyes, speaking, or moving purposefully AND
starts to breathe normally

 You become exhausted.


DISCONTINUING RESUSCITATION – DIAGNOSING
DEATH

 Absence of central pulse

 Absence of heart sounds

 Flatline – ECG

 Observe for 5 minutes

 Check for pupil response / corneal reflexes

 Involve a senior
ANY QUESTIONS?
SUMMARY

 Identifying cardiac arrest

 ALS algorithm

 Shock able / Non-shock able rhythms

 Treatment of Reversible causes – 4 H’s & 4


T’s
CARDIAC ARREST
MOULAGES
ACKNOWLEDGMENTS
 Prof. Farhat Jafri

 Prof. Tahir Husain

 Prof. Waqar Kazmi

 Dr.Wasim Siddiqui
THANK YOU ALL!

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