IMMEDIATE LIFE
SUPPORT
LEARNING OBJECTIVES
What is ILS?
Causes & Prevention of Cardiac arrest
ABCDE approach
Cardiac arrest
Cardiac Arrest moulages
WHAT IS ILS?
Provides knowledge and skills needed to treat
adult patients in cardio-respiratory arrest for
the short time before the arrival of a
resuscitation team or more experienced
assistance
Teaches the recognition and treatment of the
deteriorating patient using the ABCDE
approach.
ABCDE APPROACH
A – Airway
B – Breathing
C – Circulation
D – Disability
E - Exposure
CHAIN OF SURVIVAL
CAUSES AND PREVENTION OF
CARDIORESPIRATORY ARREST
WHY IS PREVENTION OF CARDIO-
RESPIRATORY ARREST IMPORTANT?
Once cardiac arrest occurs, fewer than 20% of
patient having an in-hospital cardiac arrest will
survive to go home.
Most in-hospital cardiac arrests are not sudden
or unpredictable events. In approximately 80%
of cases clinical signs deteriorate over the few
hours before arrest.
CHAIN OF PREVENTION
EDUCATION
How to observe patients
Recognising signs of deterioration
Using the ABCDE approach
Simple skills to stabilise the patient pending the
arrival of more experienced help
MONITORING
Patient assessment
Measurement and recording of vital signs
RECOGNITION & CALL FOR HELP
To identify patients in need of additional monitoring or
intervention (e.g Early Warning scoring system)
Protocols for summoning a response to a deteriorating
patient (clear deterioration prompts early help)
Using a structured communication tool such as SBAR or
RSVP to call for help
EARLY WARNING SCORING SYSTEM
Score 3 2 1 0 1 2 3
<45 30 15% Normal for 15% >4
Systolic BP 30%
% % down patient up 5%
<4 101- 111- >1
Heart rate (BPM) — 41-50 51-100
0 110 129 30
Respiratory rate 15- >3
— <9 — 9-14 21-29
(RPM) 20 0
<3
Temperature (°C) — — 35.0-38.4 — >38.5 —
5
AVPU — — — A V P U
ESCALATION PROTOCOL BASED ON EWS
EWS Minimal Observation Escalation
frequency
Recorder’s action Doctor’s action
3-5 4 hourly Inform nurse in charge
6 4 hourly Inform doctor Doctor to see within 1 hour
7-8 1 hourly Inform doctor Doctor to see within 30
minutes and discuss with
senior doctor and / or
Consider continuous
monitoring outreach team
>9 30 minutes Inform doctor Doctor to see within 15
minutes and discuss with
senior doctor and ICU team
Start continuous monitoring
CALL FOR HELP
SBAR RSVP
SITUATION REASON
BACKGROUND STORY
ASSESSMENT VITAL SIGNS
RECOMMENDATION PLAN
RESPONSE
Assured
Specified speed
Appropriate personnel (e.g. ICU team)
EXAMPLE
CAUSES OF DETERIORATION AND CARDIO-
RESPIRATORY ARREST
AIRWAY
BREATHING
CIRCULATION
AIRWAY OBSTRUCTION
Causes
CNS depression (loss of airway patency and
protective reflexes)
Blood, vomitus, foreign body
Trauma to face or throat
Epiglottitis
Pharyngeal swelling (e.g infection, oedema)
Laryngospasm, bronchospasm
Bronchial secretions
Blocked teacheostomy or laryngectomy
AIRWAY OBSTRUCTION (contd…)
Recognition
Conscious patient Difficulty breathing, choking,
distressed
Partial obstruction NOISY
Complete obstruction SILENT, no air
movement at mouth
Accessory muscle usage, see-saw respiration
AIRWAY OBSTRUCTION (contd…)
Treatment
Treat the cause (e.g suction)
Protect airway
airway opening manoeuvres (head tilt / chin lift
or jaw thrust)
insertion of adjuncts (e.g. oropharyngeal)
intubation
Oxygen
BREATHING PROBLEMS
Causes
Acute or Chronic
Lung disorder infection, COPD, hemo /
pneumothorax, embolism
Respiratory drive CNS depression
Respiratory effort Spinal cord damage, diseases
(myasthenia gravis, GBS), pain
BREATHING PROBLEMS (contd…)
Recognition
Conscious patient shortness of breath
Irritability, confusion, lethargy, reduced consciousness
Cyanosis – LATE sign
Indicators:
R/R
Pulse Oximetry
ABG’s
BREATHING PROBLEMS (contd…)
Treatment
Oxygen
High flow to start
Aim for 94-98% saturation on stability
COPD – 88 – 92% saturation on stability
Treat the cause
Non-invasive ventilation
Controlled ventilation (requiring intubation)
CIRCULATION PROBLEMS
Causes
Primary Heart Problems ACS, valvular, etc
Secondary Heart Problems
Respiratory arrest causing lack of oxygenation
Anemia
Septic shock
Hypothermia
CIRCULATION PROBLEMS (contd…)
Recognition
Chest pain
Shortness of breath
Syncope (fainting)
Tachycardia / bradycardia
Tachypnea
Hypotension
Poor peripheral perfusion
Altered mental status
Oliguria
CIRCULATION PROBLEMS (contd…)
Treatment
ECG
Treat the cause I/V fluids for hypotension
For ACS:
Sit patient up
Aspirin 300mg
Nitroglycerine (unless hypotensive)
Oxygen
Morphine for relief of pain
If STEMI decide for thrombolysis / PCI
ANY QUESTIONS?
SUMMARY
Causes of deterioration in patients
Importance of vital observations
Good and early communication
Rapid initiation of treatment
ABCDE APPROACH
A - AIRWAY
Assess
see-saw respiration, accessory muscle usage
cyanosis
noisy / silent or no breath sounds
depressed consciousness
Treat
Airway opening maneuvres (head tilt, chin lift / jaw
thrust)
Suction
Insertion of airway adjunct / intubation (expert only)
Oxygen
B - BREATHING
Look, Listen, Feel
R/R
Depth, chest expansion
Chest deformity
sweating, central cyanosis, accessory muscle
usage, abdominal breathing
Added sounds
Percussion + Auscultation
Tracheal position
Chest wall palpation
B – BREATHING (contd…)
Treat
Oxygen (for COPD aim for 88-92%)
Needle thoracosotomy, chest drain insertion
C - CIRCULATION
Assess
Colour and feel of hands
Capillary refill time
Pulse rate
Character of peripheral and central pulses
Blood pressure
Auscultate the heart
Conscious level
Urine output (if catheterised)
Ongoing bleeding, collections in drains
C – CIRCULATION (contd…)
Treat
Insert 14 or 16 G I/V cannula (one or more)
Take blood samples for investigations and group and
save
Rapid fluid challenge of N/saline or Hartmann’s:
If normotensive - 500 ml
If hypotensive – 1 liter
If known cardiac failure - smaller volumes (250ml) with
closer monitoring
C – CIRCULATION (contd…)
Treat
Control bleeding
Reassess and repeat fluid challenge if no
improvement
Reassess and ask for help if no improvement
(inotropes or vasopressors maybe needed)
Get ECG and follow ACS protocol if appropriate
D - DISABILITY
Assess
Review ABCs
Consciousness level
A – Alert
V – responds to Vocal stimulus
P – responds to Painful stimulus
U – Unresponsive
Or GCS
Pupils
Blood GLUCOSE
Drug induced causes
Lateral position if airway not protected
E - EXPOSURE
Full exposure of body
Respect patient’s dignity
Minimise heat loss
ALWAYS
Remain systematic in approach
First initiate treatment for identified problem,
then move to next step
Reassess and review your interventions
starting from A going to E
Don’t Ever Forget Glucose
Obtain history from patient, relative, staff,
notes
Document assessment, intervention and
responses
ANY QUESTIONS?
SUMMARY
Systematic structured ABCDE approach
Importance of treatment with identification
DEF – Glucose
Continuous re-assessments
CARDIAC ARREST
CHEST COMPRESSIONS
Correct hand position – middle of the lower half of
the sternum
High quality compressions:
Depth of 5-6 cm
Rate of 100-120 compressions per minute
Allow the chest to recoil completely after each
compression
Minimise interruptions. Plan when and why to pause
Do not feel for pulse to assess compression
effectiveness
If intubated, continue compressions uninterrupted
ADVANCED LIFE SUPPORT
Standardised approach to patient with cardio-
respiratory arrest.
Team members can predict and prepare for the next
stage in patient’s treatment
Most important interventions that improve survival
after cardiac arrest are:
Early, uninterrupted high quality chest compressions
Early defibrillation for VF / VT
Drugs and advanced airways are of secondary
importance
VENTRICULAR TACHYARDIA
VENTRICULAR FIBRILLATION
FINE VENTRICULAR FIBRILLATION
ASYSTOLE
PULSELESS ELECTRICAL ACTIVITY
ADVANCED LIFE SUPPORT - SHOCKABLE
Interval between stopping compressions and
delivering a shock must not be more than 5
seconds ideally
Resume chest compressions immediately after
a shock without checking the rhythm or a pulse
Give adrenaline 1mg (1:10000) I/V after 3 rd
shock. Repeat if arrest persists every alternate
cycle.
ADVANCED LIFE SUPPORT – SHOCKABLE (contd…)
Give amiodarone 300mg I/V after 3 rd shock.
Give further 150mg I/V if VF / VT persists.
If amiodarone is unavailable, consider 100mg
Lidocaine. Give an additional 50mg if
necessary
If rhythm is organised – check for pulse.
ADVANCED LIFE SUPPORT – NON-SHOCKABLE
Pulseless Electrical Activity: organised cardiac
‘electrical activity’ in the absence of palpable
pulses.
Give adrenaline 1mg (1:10000) I/V as soon as
intravascular access is achieved. Repeat if
arrest persists every alternate cycle.
DURING CPR
Give good quality chest compressions
Recognise and treat reversible causes
Obtain a secure airway
Obtain a vascular access
AIRWAY AND VENTILATION
Use a bag mask / supraglottic airway device
Ventilate the lungs at 10 breaths / minute
Tracheal intubation should only be attempted
by trained healthcare provider
Once intubated continue chest compressions
at a rate of 100-120 without pausing ventilation
VASCULAR ACCESS
Peripheral venous cannulation is quicker,
easier and safer.
Give flush of 20 ml of fluid post drug
administration. Elevate the extremity for 10-20
seconds.
If I/V access cannot be established, consider
intraosseous access.
REVERSIBLE CAUSES
4 H’S
Hypoxia
Hypovolaemia
Hypoglycemia / hypo- or hyper- kalemia /
hypocalcemia / acidaemia / metabolic disorders
Hypothermia
REVERSIBLE CAUSES (Contd…)
4 T’s
Tension pneumothorax
Tamponade
Toxins
Thrombosis (pulmonary or coronary thrombosis)
SIGNS OF LIFE
Respiratory effort
Movement
Check for organised rhythm
Check for pulse
If no pulse – continue CPR
CONTINUE RESUSCITATION UNTIL
Qualified / senior help arrives and takes over
The patient starts to show signs of regaining
consciousness, such as coughing, opening his
eyes, speaking, or moving purposefully AND
starts to breathe normally
You become exhausted.
DISCONTINUING RESUSCITATION – DIAGNOSING
DEATH
Absence of central pulse
Absence of heart sounds
Flatline – ECG
Observe for 5 minutes
Check for pupil response / corneal reflexes
Involve a senior
ANY QUESTIONS?
SUMMARY
Identifying cardiac arrest
ALS algorithm
Shock able / Non-shock able rhythms
Treatment of Reversible causes – 4 H’s & 4
T’s
CARDIAC ARREST
MOULAGES
ACKNOWLEDGMENTS
Prof. Farhat Jafri
Prof. Tahir Husain
Prof. Waqar Kazmi
Dr.Wasim Siddiqui
THANK YOU ALL!