1
College of Health and Medical Science
Department of Pediatrics and Child health Nursing
OVERVEIW OF PEDIATRICS SKIN PROBLEMS
By Ashenafi Z
JANUARY, 2024
01/01/2025
2 Presentation outline
Introduction
Inflammatory& allergic reactions of the skin
Bacterial skin infection
Viral skin infections
Fungal skin infection
Parasitic skin infestation
Non infectious inflammatory dermatoses
Benign tumors and skin cancer 01/01/2025
3 Morphology of skin lesions
Type of the lesion
Size of the lesion
Shape of the lesion DIAGNOSIS
Distribution and arrangement
Margination
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Primary lesions
4
o macula - spot
o Flat, change in skin color <1.0cm
o No elevation or depression
o Not identified by mere touch
o Patch ---- >1.0cm
Papule
Papula - pimple
Small solid elevation
< 0.5cm in diameter
Epidermal, dermal or 01/01/2025
combined changes
Nodule
5 Nodulus - ‘small knot’
Palpable, solid, round lesion
big papule
Blister (vesicle, bullae)
Vesicula - ‘little bladder’
Bulla - ‘bubble’
Small raised fluid filled lesion ~ 0.5cm
Vesicle < Bullae
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Pustule
6 Small pus containing swelling
Usually ass. with infection
from papules or vesicles
May develop denovo e.g. acne
Wheal
Round or flat topped red, purple papule
Disappears in hours or a day or two
Due to edema in the upper dermis
If lower----SC---Angioedema
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Plaque
7 palpable lesions >1 cm in
size and have a flat surface
Tumor
Elevated, palpable, solid mass or cystic
elevation >2 cm in diameter extends deeper
into the dermis
Cyst
Encapsulated fluid-filled or semisolid mass
in the subcutaneous tissue or dermis
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Secondary lesions
8 Erosion, ulcer
Types of tissue loses varying in their
depth and breadth
Erosion: confined to the epidermis
Ulcers: larger defect involving the dermis
and SC
Fissures
Linear crack in the skin, may extend to
dermis
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Scales
Flakes secondary to desquamated, dead
9 epithelium
Varies in color and texture
Crust
dried residue of serum, blood, or pus on skin
surface
Scar
skin mark left after healing of a wound or lesion
Keloid
hypertrophied scar tissue
Secondary to excessive collagen formation
during healing (elevated, irregular, red) 01/01/2025
Diagnostic evaluation of skin problems
1 1. History and physical examination
0 The diagnosis of a skin disorder is made chiefly by visual
inspection, but some skin disorders may require additional testing
2. Skin biopsy
Is performed to obtain tissue for microscopic examination
by scalpel excision or by a skin punch instrument that removes a
small core of tissue. Eg malignancy and exact dxs
3. Skin scraping
Tissue samples are scraped from suspected fungal lesions with a
scalpel blade moistened with oil and examined microscopically
. 01/01/2025
4. Wood’s light examination
1 Is a special lamp that produces long-wave ultraviolet rays, where it is
1 possible to differentiate epidermal from dermal lesions and hypo
pigmented and hyper pigmented lesions from normal skin.
5. Tzanck smear
The Tzanck smear is a test used to examine cells from blistering skin
conditions and The secretions from a suspected lesion are applied to a
glass slide, stained, and examined
6. Culture and sensitivity
To confirm the causative agent and drugs to which they are sensitive
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1.Inflammatory& allergic reactions of the skin
1 Dermatitis is an inflammation of the skin.
2 It is a common condition and not life-threatening or contagious.
Several types of dermatitis (eczema) Causes
Atopic dermatitis Allergies
Contact dermatitis Genetic factors
Seborrheic dermatitis Physical and mental stress
Neuro dermatitis Irritants
Stasis dermatitis
Perioral dermatitis
S/S :- redness, swelling, itching and skin lesions. 01/01/2025
Contact dermatitis
1 Acute or chronic inflammatory skin disorder
3 Characterized by well demarcated skin lesions
History of exposure to suspected offending
agent
Types:- Irritant contact dermatitis
Allergic contact dermatitis
Causes
Irritant substances Ex: Detergents,
hair metals, perfumes, strong acids &
alkalis induce irritant contact dermatitis
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S/S
o Erythematous
1
4 o Macular, papular or papulo-vesicular
eruptions,
o chiefly involving the exposed part of the
body
o Pruritus is common
Mgt
Avoid the agent
Topical steroids and if severe systemic for
a short time
Antipruritics
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Treat the complications
Atopic dermatitis
1 Is the cutaneous expression of the atopic state to environment.
5 Chronic superficial inflammation of skin characterized by pruritis
It affects 10-30% of children worldwide
2/3rd of patients have family members with
asthma, hay fever, and very dry skin
Causes
Exact cause is not known
Genetics
Activated by the immune system (IgE)
Contact with the external trigger 01/01/2025
and infection
1
6
S/S :-Pruritus, redness, papule,
thickening of skin
Exacerbations & remissions of s/s
Common over face & neck, behind
the knee & back of the hand.
Mgt:- Removal of allergen
Relief of pruritus
fluid & nutrition
Topical drugs
Systemic drugs
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Prevention of infection
Seborrheic dermatitis
1 Called dandruff in the scalp
7 Cause not fully understood
Associated factors:
Genetics
Immunosuppression
Yellowish red, often greasy, or white
dry scaling macules and papules
Mgt
Selenium sulfide shampoo
Ketoconazole shampoo
Topical steroids 01/01/2025
Acne
result from blockages in sebaceous gland follicles
1 Causes:- exactly unknown
8 Family history
Hormonal activity
Hyperactive sebaceous glands
Accumulation of dead skin cells
Types:- Acne vulgaris :- adolescent
Acne Rosacea:- uncommon < 30 yrs
S/S:- Whiteheads or blackheads of Nodules /red
lesion pustules, Scarring, Comedo
Mgt:- cleaning and removal of oils
Keep the skin clean
Antibiotics 01/01/2025
Corticosteroids
2. Bacterial skin infection
1 Boil (furuncle)
9 inflammation of hair follicles accumulation
of pus & dead tissue
Etiology:- staphylococcus auerus or
streptococcus
S/S:- Red, shiny, & swollen pus-filled lump
Tenderness, warm&/or painful
Fever & swollen lymph nodes if sever
When the lump is ready to rupture, a pointy
white or yellow central area is noticed.
Mgt:- Surgical drainage(incision & drainage)
Systemic antibiotics 01/01/2025
Vitamin A & E supply for recurring cases.
carbuncle
extension of a furuncle that has invaded several
2 follicles & large & deep seated.
0
Etiology:-usually staphylococcal infection
C/F:- painful lésion, Fever, malaise
Common sites for the lesion/thick & inelastic
skin , Back of the neck ,Shoulder
hips ,thigh
Mgt:- Extraction (surgical drainage)
o Antibiotics
o analgesics
o Topical RX – 1% GV
o Warm moist compresses 01/01/2025
o
Impetigo
2 superficial skin infection
1 More common in children and contagious
caused by either Staphylococci or
Streptococci
Two clinical syndromes
1.Non bullous
most common 70 percent
The face (especially around the nares)
commonly affected.
vesicle or pustule that quickly evolves into a
honey-colored crusted plaque. 01/01/2025
Surrounding erythema may be present.
Bullous
2 Mainly affects newborns and infants
2 Always caused by S. aurous strains that produce
exfoliative toxins.
Clear yellow fluid that subsequently becomes dark
yellow and turbid
Appear in various skin areas, esp. the buttocks
Mgt
Removal of the crusts
Antibiotics
Topical agents –GV applied 2-3x daily
Avoid contact and sharing of clothes
Complications:-AGN (PSGN),Rheumatic fever,
Cellulitis 01/01/2025
CELLULITIS
2 Inflammation of subcutaneous tissue
3 S. aureus and group A streptococci most
common etiologic agents
Preceded by a history of an antecedent
lesion
erythema, local pain, and tenderness and
variable degrees of systemic SxS
Tender and hot, vaguely demarcated lesion
with swelling of the affected limb
Regional lymph nodes may be involved.
Mgt based on purulent/ non purulent SSTIs
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Parenteral antibiotics for at least ten days
3.Viral skin infections
Herpes zoster
2 is an infection caused by the vzv
4 Common in immune-suppressed patients
painful vesicular eruption along the area of distribution
of the sensory nerves from one or more posterior
ganglia
S/S:- Pain (burning, tearing or sharply cutting)
Itching, constant aching, or deep, shooting pain.
Rashes and Blisters follow the path of the nerve
crust and Scar
Mgt:- Acyclovir
Antihistamine to reduce itching
Minimize pain
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Reduce viral shedding
Herpes simplex
Infection of the skin or mucous membrane by
2
HSV-1 and HSV-2
5 cause a wide variety of mucocutaneous
infections
produce both primary and recurrent
infections
Herpes labialisis caused usually by HSV -1,
and herpes genitalis commonly by HSV-2.
Prodromal of tingling, itching, or burning
sensation usually precedes any visible skin
changes by 24 hours
Erythema are often noted initially, followed
soon by grouped, often umbilicated vesicles 01/01/2025
Treatment :- Acyclovir
Warts
2 Non cancerous skin growths
6 Caused by direct contact with a virus called the
HPV (Human papillomavirus)
Which causes a rapid growth of cells on the
outer layer of the skin
S/S:- Small, fleshy, grainy bumps ,Flesh-
colored, white, pink
Rough to touch & usually painless
one or more tiny black dots, which are
sometimes called wart seeds
Mgt:- Salicylic acid(Podophylin)
Freezing (cryotherapy )
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Minor surgery
MOLLUSCUM CONTAGIOSUM
2 Large double stranded DNA viral infection
7 Affects children age 2-6 yrs and immunosuppressed
individuals
Spread by person to person contact
S/S:- Small raised pink bumps with dimple in center,
“umbillicated”
The face, eyelids, neck, axillae, and thighs
commonly involved
Mgt
Mostly self-limited
Immunotherapy 01/01/2025
avoid shared baths and towels
Fungal skin infection
2 Clinical forms dermatophytosis
8 (Ringworm, Tinea)
Tinea corporis (body)
Tinea capitis (scalp)
Tinea cruris ( groin & axillae)
Tinea unguium(Fungal nails)
Tinea pedis (Athlete's foot)
Diagnosis
Primarily based on the appearance &
location of the lesion.
Skin scraping 01/01/2025
Mgt
2 Topical anti fungal creams can clear the
9 condition in two weeks
o Whitfield’s ointment
o Clotrimazole1% cream
Systemic treatment
First line:- GriseofulvinV
Ketoconazole
Keep the area clean
Shampoo hair
Minimize sweat & moisture
Avoid contact 01/01/2025
Tinea versicolor(Pityriasis versicolor)
o common fungal infection of the skin
3 o interferes with the normal tanning of the skin appears as a
0 tissue-thin coating
o occurs when the fungus becomes overgrown on the skin of
hair follicles
o patches of discolored skin that grow slowly the only sign
o The patches can be various colors, including white, pink, tan
or dark brown.
o Usually affects the back, chest, neck or upper arms & can
cause mild itching.
Treatment Wash and dry the affected area,
o apply Antifungal creams, lotions or shampoos
o Ketoconazole2% cream 01/01/2025
o Flucoconazole for 2-4 wks.
4. Parasitic skin infestation
3 Scabies
1 infestation of the skin by the itch mite sarcoptes scabiei
disease of people living in substandard hygienic
conditions
spread of scabies is determined by the extent and
duration of physical contact
Children and sexual partner of an affected individual are
most at risk.
S/S:- intense pruritus, particularly at night.
Threadlike burrows are the classic signs
Preferred sites are the inter digital spaces, wrist flexors,
anterior axillary folds, ankles, buttocks, umbilicus and
belt line, groin, genitals and areolas . 01/01/2025
Mgt
3 permethrin 5% cream is standard therapy
2 Benzyl Benzoate Lotion (BBL 25%)
Sulphur (5%) applied to the entire body
Warm, soapy bath to remove the scaling
debris
All bedding and clothing should be
washed in a very hot water and dried on
hot climate
Contacts should be traced
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Pediculosis: lice infestation
They inject their digestive juices and excrement into the skin, which
3 causes severe itching
3 Serves as a vector of human disease
They depend on the host for their nourishment, feeding on human
blood approximately five times each day
disease of unwashed people or those who live in close quarters and
do not change their clothing
Types Pediculosis capitis
o Pediculosis corporis and
o Pediculosis pubis
S/S:- eggs are visible to the naked eye
Itching and scratching, Reddish brown dust
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Mgt
3
Lindane(1%,1-2oz apply for 4 mins) then
4
Wash with a shampoo/lotion containing or Pyrethrin(1%)
Permethrin1% is applied to affected areas of the skin and
to hairy areas
All articles, clothing, towels, and bedding that may have
lice or nits should be washed in hot water—at least 54°C
(130°F)
All family members and sexual contacts must be treated
personal hygiene education
01/01/2025
5.Non infectious inflammatory dermatoses
3 Psoriasis
5 Is a chronic noninfectious inflammatory and
incurable skin disease
An autoimmune-related disease
Characterized by inflammation and altered
keratinocyte proliferation
has a complex multifactorial genetic basis
Risk factors
Family history(50% pts)
medical conditions and medication
Stress Obesity
Smoking infections 01/01/2025
Injury to the skin
Classifications
3 Non pustular forms pustular forms
6 Plaque psoriasis Pustular psoriasis
Guttate psoriasis Pustolosis palmaris
Psoriatic arthritis Erythrodermic psoriasis
Signs and symptoms
Thick silvery scales
Itchy, dry, red patches and painful
Pitted, discolored, and crumbling nails
Positive Auspitz’s sign
Koebner phenomenon
The scalp, face, elbows, knees, lower back,
and genitalia are most often affected 01/01/2025
Management
Treatment is palliative and aims to slow rapid turnover
3 of the epidermis
7 Has 4-tier process
1. Topical therapy
o emollients, vitamin D analogs
o mid- to high-potency corticosteroids
2. Phototherapy
Narrow-band UVB(311 nm; NB-UVB) radiation
3. Systemic therapy
Methotrexate (0.2-0.7 mg/kg/wk
retinoids (0.5-1.0 mg/kg/day)
cyclosporine (3-5 mg/kg/day) 01/01/2025
6.Benign tumors and skin cancer
3
8 Common benign tumors in children
Epidermal inclusion cyst
Pilomarticoma
Milium
Pilar cyst
Ereptive vellus hair cysts
Syringoma
01/01/2025
skin cancer
3 Skin cancer can be classified in to two
9 Melanoma and
Non melanoma
Non melanoma is classified in to two
Basal cell carcinoma and
Squamous cell carcinoma
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40
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