Applied aspects of Tridosha Siddhanta

Applied aspects of Tridosha Siddhanta

• 1.
  Applied aspects of Tridoshasidhanta Dr.Vinodkumar M V Professor, Dept. of Samhita, Sanskrit & Sidhanta VPSV Ayurveda College Kottakkal, Kerala [email protected]
• 2.
  Tridosha Explains relatedness ofa living organism to all possible variables in and out its matrix Characters under continuous variation
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  Biological dynamics Not necessarilyfollow physical dynamics
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  Shift from Philosophyto Practice Phiolosophy, the raw material Panchabhoota, refined material extracted from raw material Tridosha, fine product from refined material Treatment, final application, based on need
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  From 2 to3  Usually Universe is explained through dichotomic expressions like  Ushna X Seeta (Agneeshomeeya)  Snigdha X Ruksha (Snehasara)  Tridosha explains Trichotomy  Shift from Dichotomy to Trichotomy is important
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  Ushna Seeta Rooksha Snigdha Pitta ? Kapha Stability Vaata Mobility Seeta+Ruksha – Vata Seeta+Snigdha- Kapha Ushna+Sngdha - Pitta Where is Ushna+Ruksha A rare, abnormal, destructive phenomenon
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  Seeta- snigdha Seeta- rooksha Ushna- snigdha
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  TRIGUNA PANCHABHOOTA TRIDOSHA Evolution Universal perspective Material perspective Biological perspective
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  Relative status inhealth and diseases Dosha – They are easily disturbed and put disturbance to the system Dhatu – Normally they support the biological system Mala – They sometimes contaminate the body Sthoona – Behave like three pillars, to support the biological system
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  TRIDOSHA – isit ‘DRAVYA’ or not?
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  THE RECIPROCAL DEFINITION OFDRAVYA WHEREVER THERE IS QUALITY & ACTIVITY THERE SHOULD BE AN UNDERLYING DRAVYA
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  IF ‘DRAVYA’ ISNOT PERCEIVABLE GO FOR ITS QUALITIES AND ACTIVITIES Is sugar directly perceivable it the tea?
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  Tridosha - Definitions NOT Interms of structure BUT Qualities and activities
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  Gunas (Qualities)  Gunasnot for description but for APPLICATION  Main basis to apply saamaanya-visesha sidhaanta  Exclusively Gurvaadiguna;  Exceptions - Pitta & Kapha (visra & mritsna)  There is a cause-effect relation between Guna & Karma
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  Dormant & DominantGunas  Almost all the functions of Doshas are expressions of underlying Gunas  All the Gunas of Doshas are not equally active in all the functions  Eg: Normal functions of Vata (Utsaha-achwasa-niswasa etc.) predominate with Calaguna only  Then where are the other Gunas?  So, Gunas should be understood distinctively as dominant Gunas & dormant Gunas
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  Rule - 1 Guna dormant in normal state may become dominant in abnormal state  Eg: Vata – Normal function – Utsaha etc – dominant Guna – Cala  Vata – increased presence (Vriddhilakshana)  Kaarsya – Laghu  Karshnya – Ruksha  Ushnakamitwa – Seeta  Kampa – Cala  Sakritgraha – Ruksha, Seeta etc. Different Gunas exhibit their functions
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  KOSHT’HA NORMAL INCREASE CALA ROOKSHA NORMAL PERISTALTIC MOVEMENT CONSTIPATION For Vata
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  Rule - 2 Quality dormant in one activity may become dominant in another activity in the same state of Dosha  See the following examples for Vata SANGAM STAMBHAM S’EETA KAMPAMCHALA REDUCED MOVEMENT HYPER MOBILITY
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  Rule - 3 Quality dominant in one site/situation may be dormant in another site/situation Site VAATA PITTA KAPHA AGNI Vishamaagni due to CHALA (ANAVASTHITATWA) Teekshna due to USHNA TEEKSHNA Manda due to GURU MANDA KOSHT’HA Kroora due to ROOKSHA S’EETA Mridu due to DRAVA SARA Madhyama due to SEETA SNIGDHA
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  PITTA TEEKSHNA PRAKAASA USHNA SNEHA Pakti Ooshma Darsana Prabha Medha Dhee Saurya Tanumaardava Paachaka Ranjaka Bhraajaka Aalochaka Saadhaka
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  25042011 21RoTP TPRA MINDSharpness Mobility Stabilty Easiness Receptivity Paatava Utsaaha Medha Dhee Smriti Kshama Descrimination Judgement Retention Memory Vaata Kapha Pitta
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  Variations of Dosha (Doshabheda) Sites (Sthaana)  Functions (Karma)  Stage (Avasthaa)  Cause (Hetu)  Manifestation (Aakriti)  Management (Saadhana)  Combination (Samyoga) Ref: Doshabhedeeya A H Soo 12 (Sarvangasundara)
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  Chief locations  Relatedto feeding, processing & evacuation  Related to sites of production in the gut Importance of gut in biology
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  INLET OUTLET VACCUM AT THE BOTTOM PROCESSING Heaviness Lightness
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  MAN
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  Sites – clinicalimportance  Includes dhaatu, kosht’ha, indriya etc.  Clinical - Compare with rogamaarga  Vaata – mostly madhyama  Kapha-pitta – mostly – saakha,kosht’ha  Related to prognosis – tulya/atulyadooshyata  Grouping of syndrome  eg: aamavaata – “sleshmasthaanam pradhaavati”  Sleshmaas’ayaanaam soonyatvam
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  Functional varieties (karmabheda) Five types of doshas  Classification - on the basis of function
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  Functional sites  Mainrepository (moolasthaana) & functional territories (gocharasthaana)  Almost totally different from structural sites  Function more important than site  Physiological importance stressed initially  Pathologically - Becomes more important in the context of aavarana  Explains the harmonious co-existence of doshas
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  Stage (avastha)  Accumulation(chaya)  Circulation (prakopa)  Normalization (pras’ama)  Variants – s’eeta and ushna  Explains seasonal phenomena  Practically explainable
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  Varieties of cause(hetubheda)  Three types of causative agents  Includes food, physical activities, mental activities, facultative activities, seasonal variations etc.  Not dosha specific  Not disease specific  Only headings
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  Varieties of management (saadhanabheda) Snigdhopas’aya – Vaata, vaata-pitta  Rookshopas’aya – kapha, kapha-pitta
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  Varieties of association(samyogabheda)  Samsarga  Sannipaata  Then what is anubandha…?  Because any two of the doshas have some common features  In the process of disease one dosha may take up another dosha with it
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  Dosha vikriti  Decrease(Kshaya)  Increase (vriddhi)  Signature features (Aatmaroopa)  Morbid activities (Kupitakarma)  Independent diseases (naanaatmajavyaadhi)  Dependent diseases (saamaanyajavyaadhi)
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  Increase and decreaseof doshas are understood through increase of some or other qualities
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  Symptoms of Increase(vriddhilakshana)  General indicators of particular dosha  Students’ guideline for dosha  Not a clinician’s guideline  Not disease-specific  But, sometimes help for differential diagnosis  Important during an ‘identity crisis’
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  Clinical importance  Notsuggesting the process of disease but, indicating doshaja variety of a disease or to identify anubandhadosha  Eg: kshut, trit (pitta)  Some times very important in designing treatment  Trishna in jvara  Alpanidrata in depression
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  Signature features (aatmaroopa)* Mentioned in Carakasamhita  Sure indicators of particular dosha  सर्वेष्र्वपि खल्र्वेतेषु र्वातपर्वकारेषूक्तेष्र्वन्येषु चानुक्तेषु र्वायोररदमात्मरूिमिररणामम कममणश्च स्र्वलक्षणं, यदुिलभ्य तदर्वयर्वं र्वा पर्वमुक्तसन्देहा र्वातपर्वकारमेर्वाध्यर्वस्यन्न्त कु शलााः (C.S.Su 20/12)  Responsible for different activities when localized in tissues  Guna-karma relationship between aatmaroopa-prakupitakarma
• 38.
  Signature features (aatmaroopa) Vaata Raukshya Saitya Laaghava Vaisadya Gati Amoortatva Anavasthitatva Pitta Aushnya Taikshnya Dravatva Anatisneha Suklaarunavarjya- varna Visragandha Katukaamlarasa Saratva Kapha Sneha Saitya Sauklya Gaurava Maadhurya Sthairya Paichilya Maartsnya Ref:Carakasamhita Sutrasthana 20/12,14,16
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  Morbid activities (prakupitakarma)  Indicators of pathological processes at different sites of action  Basic understanding of mechanism of dosha action  Should be understood in comparison with site of action  Related to sthaanasamsraya of dosha in different sites  Dhaatugatha avastha of doshas can be taken as a simple model
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  Pathogenesis Aatmaroopa Dosha Dooshya Prakupitakarma sthaanasamsraya Disease Saamaanyaja naanaatmaja
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  Diseases – twotypes  Aatmaroopa of a single dosha expressed at a site leads to Naanaatmajavyaadhi  nakhabheda - raukshya at nakha –  Anavasthitachittatva – anavasthitatva of manas  Davathu – aushnya of indriyas  Aatmaroopa of more than one dosha expressed at a site leads to Saamaanyayaadhi
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  Vaata – prakupitakarma Ref:A H Sutra
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  Sramsa/Bhrams’a  Caused bylaghutva & chalatva  Loosening /dislocation/displacement  Loosening – muscle tone/ flaccidity  Dislocation – joint  Muscle dislocation – prolapses, intussusception, ptosis, hernia etc.  Dislocation – in aasayaapaksrshagati, atisaara (visramsayati abdhaatum)
• 44.
  Vyaasa - dilation Due to predominance of aakaas’a & lightness  Production of avakaas’a in dhaatukshaya  Mechanism of hypertrophy  Dilation of lumens – aneurysm, cardiomegaly, brochiectasis etc.  In the sampraapti of raajayakshma – “ativivritya vaa”
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  Svaapa  “kriyaasu achaitanyam” Sensory loss
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  Saada  Functional insufficiency(kriyaasu asamartha)  Deficiencies of different factors Eg: insulin deficiency (type II)
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  Sankocha & Sanga– Partial and complete obstruction  Due to seeta, rooksha qualities  Mostly of a lumen  Mostly mechanical  Otherwise called rodham  GI tact - udara, gunma, udaavarta, ruddhapadakaamala  Respiratory tract – swaasa, kaasa  Mootravahasrotas – mootraaghaata, asmari  Circulatory system – atherosclerosis etc.
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  Vartana  Pindeekarana –mass formation  Gunma, Space occupying lesions etc.
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   Kampa  dueto chalatva  tremor  Paarushya  due to kharatva  harshness (of voice)
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   Soushirya  dueto saukshmya  porosity (mainly in bone tissue)  S’osha  due to rooksha & laghu  wasting, dystrophy, degeneration  Spandana  Due to chalatva  Fasciculations, involuntar y movements
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  Stambha  Due toseeta+rooksha  Solid medium - Rigidity, stiffness  Liquid medium – arrest of flow - (like in coagulation)  Malastambha
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  sosha Bhrams’a Paarushya vyaasa sanga
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  Diseases Naanaatmaja Absence of otherdoshas in history, clinical examination or anupas’aya-anupas’aya Saamaanyaja Presence of other doshas in history, clinical examination or upas’aya- anupas’aya
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  Independent diseases (naanaatmajavyaadhi)  Independentof other morbid factors  Mostly localized diseases  Minimum systemic involvement/pathogenesis  Linear correlation with aatmaroopa
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  General diseases (saamaanyajavyaadhi) Involvement of more morbid factors  Process of pathogenesis well identified  Involvement of co-morbid factors like srotas, agni, aama, ojus etc.
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  Conversion  Some diseasesmay have naanaatmaja and saamaanyaja stages Eg: Gridhrasi, pakshaaghaata etc.  Sometimes, at a particular stage of treatment a saamaanyaja disease can turn naanaatmaja (keveladoshaavastha)  Most of the saamaanyajavyaadhi can not become naanaatmaja Eg: swaasa, udara, raajayakshma etc.
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  Paaka Sruti, kleda Sanga, sankocha Vyaasa Saushirya Scar, lossof elasticity (kaathinya)
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  Guna Aatmaroopa Prakupita- karma DooshyaVyaadhi Naanaatmajam Saamaanyam
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  Mutual interaction  Aama Aavarana
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  Exceptions  Vikritivishamasamavaaya
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  Beyond tridosha  Rishta Bhootagraha
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  TRIDOSHA – POSSIBLEVARIATIONS Normal Prakriti Normal variation Chaya Prakopa
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  TRIDOSHA – POSSIBLEVARIATIONS Normal Prakriti Normal variation Chaya Prakopa
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  COMBINATIONS SAMSARGA SANNIPAATA
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  SIX COMBINATIONS OFSAMSARGA
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  TEN COMBINATIONS OFSANNIPAATA
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  TEN COMBINATIONS OFSANNIPAATASIX COMBINATIONS OF SAMSARGA
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  THANK YOU