Sub154410 frozen shoulder

International Journal of Science and Research (IJSR)
ISSN (Online): 2319-7064
Index Copernicus Value (2013): 6.14 | Impact Factor (2013): 4.438
Volume 4 Issue 5, May 2015
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Licensed Under Creative Commons Attribution CC BY
Different Mobilization Technique in Management
of Frozen Shoulder
Dr. Jayanta Nath (MPT, Ph.D Scholar)1
1
SSUHS, Department of orthopaedics GMC&H, Assam, India
Physiotherapist at Jugijan Model Hospital (Govt of Assam), Industrial Area, P/O Mangaldai, Dist: Darrang, Assam, PIN: 784125, India
Abstract: Adhesive capsulitis also known as frozen shoulder, is a condition characterized by pain and significant loss of both active
range of motion (AROM) and passive range of motion (PROM) of the shoulder. Frozen shoulder usually affects patients aged 40-70,
with females affected more than males, and no predilection for race. There is a higher incidence of frozen shoulder among patients
with diabetes (10-20%), compared with the general population (2-5%). There is an even greater incidence among patients with insulin
dependent diabetes (36%), with increased frequency of bilateral shoulder involvement.8
This paper reviews the various mobilization
technique like Midrange mobilization (MRM), endrange mobilization (ERM), and mobilization with movement (MWM) by Maitland,
Kaltenborn, and Mulligan and other soft tissue technique like myofascial release, Active Release Therapy (ART), for management of
patients with frozen shoulder.
Keywords: Frozen shoulder, mobilization technique, pathology of frozen shoulder
1.Introduction
Adhesive capsulitis, also known as frozen shoulder, is a
condition characterized by pain and significant loss of both
active range of motion (AROM) and passive range of motion
(PROM) of the shoulder. The term “Periarthritis” first
described by a French doctor ES Duplay in 1872. The term
“frozen shoulder” was first introduced by Codman in 1934.
The peak age is 56, and the condition occurs slightly more
often in women than men. In 6-17% of patients, the other
shoulder becomes affected, usually within five years, and
after the first has resolved. The non-dominant shoulder is
slightly more likely to be affected.While many classification
systems are proposed in the literature, frozen shoulder is
most commonly classified as either primary or secondary.
Primary frozen shoulder is idiopathic in nature, and
radiographs appear normal. Secondary frozen shoulder
develops due to some disease process, which can further be
classified as systemic, extrinsic, or intrinsic. Systemic
secondary frozen shoulder develops due to underlying
systemic connective tissue disease processes, and causes
include diabetes mellitus, hypo- or hyperthyroidism,
hypoadrenalism. Extrinsic secondary frozen shoulder occurs
from pathology not related to the shoulder, such as
cardiopulmonary disease, CVA, cervical disc pathology,
humeral fracture, and Parkinsons. Intrinsic secondary frozen
shoulder results from known shoulder pathology, including
but not limited to rotator cuff tendinopathy, GH arthropathy,
and AC arthropathy.3
Frozen shoulder usually affects patients aged 40-70, with
females affected more than males, and no predilection for
race. There is a higher incidence of frozen shoulder among
patients with diabetes (10-20%), compared with the general
population (2-5%). There is an even greater incidence among
patients with insulin dependent diabetes (36%), with
increased frequency of bilateral shoulder involvement.8
While the etiology of frozen shoulder remains unclear,
several studies have found that patients with frozen shoulder
had both chronic inflammatory cells and fibroblast cells,
indicating the presence of both an inflammatory process and
fibrosis3
. Frozen shoulder typically lasts 12 to 18 months
with a cycle of 3 clinical stages, the freezing, frozen and
thawing stages. These stages last on average 6 months, but
the time frames are variable. The freezing stage is also
known as the painful inflammatory phase. Patients present
with constant shoulder pain and range of motion (ROM)
limitations in a capsular pattern (external rotation (ER)>
abduction (ABD)> flexion (FLX)> and internal rotation
(IR)). In the second phase, the frozen or stiff phase, the pain
progressively decreases as does shoulder motion and
individuals commonly experience increased restrictions in
function. In the last phase, the thawing phase, patients
gradually regain shoulder movement and experience
progressively less discomfort 11
.
Three phases of clinical presentation
Painful freezing phase
Duration 10-36 weeks. Pain and stiffness around the shoulder
with no history of injury. A nagging constant pain is worse at
night, with little response to non-steroidal anti-inflammatory
drugs
Adhesive phase
Occurs at 4-12 months. The pain gradually subsides but
stiffness remains. Pain is apparent only at the extremes of
movement. Gross reduction of glenohumeral movements,
with near total obliteration of external rotation
Resolution phase
Takes 12-42 months. Follows the adhesive phase with
spontaneous improvement in the range of movement. Mean
duration from onset of frozen shoulder to the greatest
resolution is over 30 months.
Paper ID: SUB154410 1285

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2.Pathogenesis
The aetiology of frozen shoulder remains unclear. The
disease process particularly affects the anterosuperior joint
capsule and the coracohumeral ligament.5
Arthroscopy shows
a small joint with loss of the axillary fold and tight anterior
capsule, mild or moderate synovitis, and no adhesions.6
Neviaser and Neviaser have described an arthroscopic four
stage classification for the frozen shoulder,5
and Hannafin et
al have described a correlation between the arthroscopic
stage, the clinical examination, and the histological
appearance of the tissues. Disagreement prevails about
whether the underlying pathological process is an
inflammatory condition, a fibrosing condition,1
or even an
algoneurodystrophic process. Evidence shows a synovial
inflammation with subsequent reactive capsular fibrosis.A
dense matrix of type I and type III collagen is laid down by
fibroblasts and myofibroblasts in the joint capsule.
Subsequently, this tissue contracts. Increased growth factors,
cytokines, and expression of matrix metalloproteinases in
capsular biopsy specimens obtained from patients with
primary and secondary frozen shoulder indicate that these are
involved in the inflammatory and fibrotic cascades seen in
frozen shoulder. Cytokines and growth factors are involved
in the initiation and termination of repair processes in
musculoskeletal tissues through regulating fibroblasts, and
the remodelling process is controlled by matrix
metalloproteinases and their inhibitors. An association
between frozen shoulder and Dupuytren’s disease has been
identified,1,12
and this may be related to matrix
metalloproteinase inhibitors.
3.Treatment point of view
Recent evidence has not been able to conclude which
treatment technique, whether physical therapy, home exercise
program, cortisone injection, manipulation, or surgery, is
most effective.10
Therefore the decision to begin and
continue with formal physical therapy should involve input
from the physician, the patient preference, and physical
therapist after initial evaluation. Patients who may benefit
more from formal physical therapy include those with higher
disability levels, higher anxiety levels, lower educational
levels, and those who have less social support.3
While
evidence regarding the use of intra-articular cortisone
injections is conflicting as well, some studies do indicate they
provide better short-term (4-6 week) pain reduction than
other forms of treatment 9.
Given this information, if this is
not offered to patients before referral to physical therapy and
they do not demonstrate progress within 3-6 weeks, referral
for evaluation for an injection should be considered.3
Prognosis:
Adhesive capsulitis can last 12 to 18 months, with 3 distinct
phases. The first phase can last 2-9 months, the second phase
4-12 months and the last phase, the thawing phase, from 6-9
months.
Although the pathogenesis of FSS(Frozen shoulder
syndrome) is unknown, several authors have proposed that
impaired shoulder movements are related to shoulder capsule
adhesions, contracted soft tissues, and adherent axillary
recess. Cyriax suggested that tightness in a joint capsule
would result in a pattern of proportional motion restriction (a
shoulder capsular pattern in which external rotation would be
more limited than abduction, which would be more limited
than internal rotation). Based on the absence of a significant
correlation between jointspace capacity and restricted
shoulder ROM, contracted soft tissue around the shoulder
may be related to restricted shoulder ROM. Vermeulen and
colleagues indicated that adherent axillary recess hinders
humeral head mobility, resulting in diminished mobility of
the shoulder. Furthermore, they documented that abnormal
scapular motion existed in patients with FSS despite
improvement in glenohumeral motion following a 3-month
period of physical therapy intervention. Apparently, impaired
shoulder movements affect function. In longitudinal follow-
up studies lasting from 6 months to 2 years, significant
numbers of patients with FSS demonstrated moderate
functional deficits. To regain the normal extensibility of the
shoulder capsule and tight soft tissues, passive stretching of
the shoulder capsule and soft tissues by means of
mobilization techniques has been recommended, but limited
data supporting the use of these techniques are available.
Midrange mobilization (MRM), endrange mobilization
(ERM), and mobilization with movement (MWM) techniques
have been advocated by Maitland, Kaltenborn, and Mulligan,
but they did not base their suggestions on research.
Additionally, few studies have described the use of these
techniques in patients with FSS.
4.Various Mobilization Technique Option
Mid-Range Mobilization
An MRM technique was performed on the involved shoulder,
as described by Maitland and Kaltenborn. With the subject in
a relaxed supine position, the humerus was moved to the
resting position (40° of abduction). While the humerus was
held in this position, 10 to 15 repetitions of the mobilization
techniques were applied.
End-Range Mobilization
In addition to the MRM technique, ERM has been
recommended. The intent of ERM was not only to restore
joint play but also to stretch contracted periarticular
structures. We used the techniques described by Vermeulen
et al and Maitland as follows. At the start of each
intervention session, the physical therapist examined the
subject’s ROM to obtain information about the endrange
position and the end-feel of the glenohumeral joint. Then, the
therapist’s hands were placed close to the glenohumeral joint,
and the humerus was brought into a position of maximal
range in different directions. Ten to 15 repetitions of
intensive mobilization techniques, varying the plane of
elevation or varying the degree of rotation in the endrange
position, were applied.
Mobilization With Movement
The use of MWM for peripheral joints was developed by
Mulligan. This technique combines a sustained application of
a manual technique “gliding” force to a joint with concurrent
physiologic (osteo-kinematic) motion of the joint, either

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actively performed by the subject or passively performed by
the therapist. The manual force, or mobilization, is
theoretically intended to cause repositioning of bone
positional faults. The intent of MWM is to restore pain-free
motion at joints that have painful limitation of range of
movement.
The MWM technique was performed on the involved
shoulder as described by Mulligan. With the subject in a
relaxed sitting position, a belt was placed around the head of
the humerus to glide the humerus head appropriately, as the
therapist’s hand was used over the appropriate aspect of the
head of the humerus. A counter pressure also was applied to
the scapula with the therapist’s other hand. The glide was
sustained during slow active shoulder movements to the end
of the pain-free range and released after return to the starting
position. Three sets of 10 repetitions were applied, with 1
minute between sets.
Scapular mobilization
Subjects lay on their sound side on the bed. The therapist
stood before the patient’s affected shoulder, placing the index
finger of one hand under the medial scapular border, the
other hand grasping the superior border of the scapula. The
scapula was moved superiorly and inferiorly for superior and
inferior glide, and then the scapula was rotated upward and
downward for scapular rotation. Additionally, the
physiotherapist put the ulnar fingers under the medial
scapular border and distracted the scapula from the thorax.
These patterns were chosen to increase scapular posterior tilt.
Ten sets of 10 repetitions were applied, with rest intervals of
30 s between sets (Wooden, 2001; Surenkok et al., 2009).
Maitland mobilization is a widely used therapeutic technique
used to treat various intra articular and periarticular
disorders. Grades I and II of Maitland mobilization
techniques are primarily used for treating joints limited by
pain. The oscillation may have an inhibitory effect on the
perception of painful stimuli by repetitively stimulating
mechanoreceptors that block nociceptive pathways at the
spinal cord or brain stem levels. These nonstretch motions
help to move synovial fluid to improve nutrition to the
cartilage whereas Grades III and IV are primarily used as
stretching manoeuvres2
.Appropriate selection of
mobilization technique for treatment can only take place after
a thorough assessment and examination.
Application of Maitland Mobilisation 2
Passive Accessory Movements
The following passive accessory movements were tested and
treated accordingly
Gleno-Humeral Joint:
· Antero –Posterior glide
· Postero – Anterior glide
· Caudal glide
· Distraction or Lateral glide
Sterno – Clavicular Joint:
· Antero – Posterior glide
· Postero – Anterior glide
· Caudal glide
· Cephalad glide
Acromio – Clavicular Joint
· Antero – Posterior glide
· Posterior – Anterior glide
· Caudal glide
· Cephalad glide
Passive Physiological Movements
The following passive physiological movements were tested
and treated accordingly
· Flexion
· Extension
· Abduction
· Internal and External rotation
MWM GH joint in abduction
Mobilizations with movements integrate well with muscle
imbalance correction of these movement abnormalities. If the
patient’s problematic movement is abduction a corrective AP
or longitudinal caudad glide on the head of the humerus can
be sustained while the patient actively abducts the arm. The
therapist’s opposite hand fixates the scapula so that the glide
of the humerus is relative to the scapula. It is important to
ensure that the AP glide is applied at right angles to the plane
of the glenohumeral joint. The resulting movement must be
pain-free. The patient can also be encouraged to activate
specific muscles. For example, once a patient has been taught
to activate a weak trapezius in isolation, the abduction MWM
can be performed while encouraging correct recruitment of
this muscle through the movement pattern.Pain-free
repetitions ensure sufficient corrective afferent input from the
joint receptors and resulting reflex changes in muscle
recruitment. A home programme of appropriate correct
movement patterns (Sahrmann SA 2002) with the addition of
taping will ensure prolonged and automatic pattern
correction.
MWM GH joint in flexion
Similar glides can be applied if glenohumeral flexion is the
symptomatic movement. A belt is usually used to glide the
humeral head backward in the treatment plane, as the use of
the therapist’s hand over the anterior aspect of the head of the
humerus will block the movement. A counter pressure is
applied to the scapula from behind. It is essential that the belt
is maintained at right angles to the plane of the joint. The belt
position on the therapist should be lower than the
glenohumeral joint; this ensures that the belt does not elevate
the humeral head and impinge on superior structures. The
belt and therapist position must be altered so that when the
glide is applied the flexion is facilitated.
5.Electrical Stimulation Technique Option
Shoulder Flexion & Abduction(Dr. Lucinda Baker, PT,
PhD, Electrode Placement &Functional Movement)
Electrode Placement & Functional Movement:
Electrode placements for shoulder flexion and abduction can
be seen here with one electrode over the anterior deltoid and

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the other over the middle deltoid. The acromion is marked
with the marker. In our first placement the negative electrode
of the asymmetric waveform is placed over the anterior
deltoid and you can see the resultant stimulation is one of
dominantly flexion. The therapist is now putting the
stimulator on pause and he is going to switch the polarity so
that the middle deltoid electrode is now the negative
electrode and the anterior deltoid will become the positive
electrode. You can see the resultant stimulated contraction is
one of dominant abduction. Placing the stimulator on pause,
the electrode leads are being disconnected. A second
stimulator that is programmed with a symmetric biphasic
waveform is being connected. And now as the therapist
increases the amplitude you will notice that both electrodes,
anterior and middle deltoid, are equally excitable resulting in
a motion that would be referred to as scaption, a combination
of flexion and abduction
pulse width: 300μsec
hz: 35
waveform: Asymmetric/Symmetric
stimulation grade: N/A
6.Chiropractic Management Option
Management technique: medical(ref: Chiropractic
management of shoulder pain and dysfunction of myofascial
origin using ischemic compression techniques. Guy Hains,
DC. J Can Chiropr Assoc 2002; 46(3)). Green recently
carried out a systematic review of the 31 clinical trials
investigating the effectiveness of various therapeutic
interventions for shoulder pain. These included anti
inflammatory medications, intra-articular cortisone
injections, physiotherapy, manipulation under anesthesia,
hydrodilation and surgery. According to Green, only sub-
acromial cortisone injections were found to be more effective
than placebo to increase abduction. This led Green to
conclude that there was little scientific evidence to support
the effectiveness of many of the most commonly used
medical therapies for shoulder pain. Similarly, Hanten et al.
Posited that, although ice, heat, ultrasound and massage are
routinely used for temporary relief for patients with trigger
points, there are no controlled studies that support their use
to decrease pain in symptomatic patients. Roubal et al.
reported on their results for patients experiencing adhesive
capsulitis using glenohumeral gliding manipulation under
anesthesia. In that study, 8 patients (4 men and 4 women
ranging in age from 31 to 55 years) with symptoms of
adhesive capsulitis from 3 to 16 months duration underwent
interscalene brachial nerve block and were immediately sent
for manipulation by a physiotherapist. The authors reported
that all patients experienced improvement in shoulder
flexion, abduction, external and internal rotation, as well as
overhead activities, dressing activities, and hair care. In
addition, patients prior to manipulation reported positive
impingement signs. None of these patients had positive
impingement signs postmanipulation and at the time of
discharge. Unfortunately, the authors did not comment on
whether or not patients experienced any adverse effects as the
result of the anesthesia. Management techniques: chiropractic
Favoring techniques that are low-tech, non-invasive and
hands-on, chiropractors typically use cryotherapy,
mobilization, manipulation and soft tissue techniques for the
management of disorders of the spine and peripheral joints.
Among the most popular method of treatment of myofascial
pain syndromes is ischemic compression. This approach, also
known as Pennel's technique, Nimmo technique, trigger point
therapy or acupressure has been used by chiropractors and
other manual therapists for at least 40 years. ' According to
the 2000 Job Analysis of the National Board of Chiropractic
Examiners, over 90% of chiropractors use trigger point
therapy for passive adjustive care, 68% use acupressure, and
40% report using NIMMO or Receptor tonus technique. It
should also be mentioned chiropractors often provide patient
education, lifestyle modifications and ergonometric
suggestions to augment the care administered in office.
Several studies have demonstrated that inactivation of TPs or
TSs often alleviates the pain the patient is experiencing
immediately, although some studies suggest that these results
may last for up to 2 years. Travel and Simons are among the
leading experts in the area of trigger point therapy, and they
have described the characteristic pain pattern associated with
different muscle groups. Active trigger points in the
supraspinatus muscle, for example, often produce a deep pain
in the medial region of the deltoid. Trigger points of the
infraspinatus muscle often cause pain in the anterior region of
the deltoid, and TPs of the teres minor muscle often lead to
pain in the posterior deltoid. Trigger points originating from
deltoid muscle itself often result in local pain. Leahy has
provided a slightly different method to treat myofascial
symptoms, especially those causing mechanical compromise
of peripheral nerves.First described as a myofascial release
technique, Leahy now refers to his soft therapy method as
Active Release Therapy (ART). The distinguishing feature of
ART is that, unlike other myofascial techniques, ART
requires the patient to perform particular actions while the
practitioner applies a static pressure along the length of the
affected muscle. In one study, Leahy et al. reported that this
technique is very effective for peripheral nerve entrapment
conditions such as carpal tunnel and thoracic outlet
syndromes. The only reported side effect is considerable pain
or discomfort that may exceed the pain tolerance of some
patients. Example of ischemic compression technique During
clinical-practice, the author has found it to be most beneficial
to exam the entire shoulder complex, and to provide ischemic
compression to any trigger points elicited. The author relies
on both his palpatory skills and patient reaction to static
pressure in order to identify the muscle groups in need of
treatment, and has developed specific protocols. According
to the author, the anatomical locations mentioned below are
the focus of most of the major irritation points in shoulder
pain. Once the TGs and TSs are localized only these points
are treated at each visit.
1. Supraspinatus (Figure 2): While prone, the patient's arm
is flexed, pronated and placed on his or her head. The
patient's arm is allowed to rest on the practitioner's knee. The
practitioner then applies digital pressure along the posterior
aspect of the clavicle, with special attention given to elicited
trigger points.
2. Deltoid (Figure 3): The patient's arm is placed in the same
position as it is for the supraspinatus. The practitioner

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applied digital pressure along the entire surface of the
deltoid.
3. Teres minor (Figure 4): While prone, the patient's arm is
flexed and braced along the side of his or her body. Using his
or her thigh, the practitioner can brace the patient's arm in
this position while applying digital pressure along the
inferolateral aspect of the scapula.
4. Infraspinatus (Figure 5): The patient's arm is placed in
the same position as it is for treatment of the teres minor
muscle. The practitioner applies pressure along the region
below the spine of the scapula.
5. The coracoid process (Figure 6): The patient is placed in
supination, the arms along the side of the body. The therapist
uses his thigh to maintain pressure on the patient's arm in
order to ensure that the arm is fully relaxed. This position is
the same for the examination of the whole front part of the
shoulder.
6. The tendon of the long head of the biceps (Figure 6):
The tendon originates from the upper edge of glenoid cavity
and transverse anterior to the head of the humerus along the
bicipital groove. TSs are often located along that tendon over
a length of 2 to 3 centimeters. During each of these
maneuvers, the practitioner applies digital pressure to areas
of elicited pain for between 5 and 15 seconds. The
practitioner should begin with light, firm pressure and
gradually increase it until the pressure reaches the patient's
maximum pain tolerance. Pressure should be evenly applied
and only once to each trigger point each treatment session.
Elicited pain to the area of the chief complaint confirms the
diagnosis. In general, the duration of applied digital pressure
varies inversely with the number of elicited trigger points.
For example, if many trigger points are elicited, each should
be held for less time than if few trigger points were found.
This is because there are limits to what a patient can endure
in any treatment session. Leahy recommends treatment
schedules not exceed every other day to permit tissue healing
between appointments. Treatments are continued until trigger
points are no longer elicited with digital palpation. In the
author's experience, this may require in chronic cases 15 to
30 treatments. Besides minor pain or tenderness of limited
duration in the area treated, no serious side effects are
associated with this type of therapy. In addition, the
application of ischemic compression for myofascial trigger
points is not very strenuous on the practioner.
7.Conclusions
Frozen shoulder is a common, sometimes painful, but
ultimately self limiting, condition that is usually managed in
the primary care setting with a combination of analgesics,
injections, and physiotherapy. Formal investigations are
usually normal, and the diagnosis is essentially clinical. Most
cases can be managed in the primary care setting. Educating
patients plays an important part in the management of the
condition. A minority of patients require referral to an
orthopaedic specialist, where manipulation under anaesthesia
is the most common method of treatment. Arthroscopic
surgical release has proved itself to be useful in refractory
cases. Irrespective of the treatment given, a high proportion
of patients with frozen shoulder do not regain a full range of
motion.
Figure 1: Arthroscope view of a shoulder with synovitis
Figure 2: Supraspinatus
Figure 3: Deltoid

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Figure 4: Teres Minor ( Posterior view of shoulder)
Figure 5: Infraspinatus
Figure 6: Anterior view of shoulder ( Long head of biceps)
References
[1] Bunker TD Anthony PP. The pathology of frozen
shoulder. A Dupuytren-like disease. J Bone Joint Surg
Br .1995;77:677-83.
[2] G. D. Maitland. Peripheral Manipulation.3rd
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Butterworth- Heinemann; 1991.
[3] Kelley MJ, McClure PW, Leggin BG. Frozen Shoulder:
Evidence and a Proposed Model Guiding Rehabilitation.
Journal of Orthopaedic and Sports Physical Therapy.
February 2009;39(2):135-136-148.
[4] Kissner C, Colby LA. Therapeutic Exercise:
Foundations and Techniques. 3rd ed. Philadelphia: F.A.
Davis Company:278-280.
[5] Neviaser RJ, Neviaser TJ. The frozen shoulder:
Diagnosis and management. Clin Orthop Relat Res.
1987;(223):59-64.
[6] Ogilvie-Harris DJ, Wiley A. Arthroscopic surgery of the
shoulder: a general appraisal. J Bone Joint Surg (B)
1986;68-B:201-7.
[7] Ozaki J, Kakagawa Y, Sakurai G, Tamai S Recalcitrant.
chronic adhesive capsulitis of the shoulder. Role of
contracture of the coracohumeral ligament and rotator
interval in pathogenesis and treatment. J Bone Joint Surg
1989;71(A):1511-5.
[8] Pearsall AI, MD. Adhesive Capsulitis. eMedicine. 2008.
Accessed 06/01/09.
[9] Ryans I, Montogmery A, Galway R, Kernohan WG,
McKane R. A randomized controlled trial of intra-
articular triamcinolone and/or physiotherapy in shoulder
capsulitis. Rheum.2005;44(4):529--535.
[10]Rookmoneea M et al. The effectiveness of interventions
in the management of patients with primary frozen
shoulder. The Journal of Bone S Joint Surgery (Br).
2010;92-B(9):1267--1272.
[11]Saidoff DC, McDonough AL. Critical Pathways in
Therapeutic Intervention: Extremities and Spine. St.
Louis: Mosby; 2002:134-144.
[12]Smith SP, Devaraj VS, Bunker TD. The association
between frozen shoulder and Dupuytren’s disease. J
Shoulder Elbow Surg.2001;10:149-51.
Author Profile
Dr.Jayanta Nath is currently working as a
physiotherapist at Jugijan Model Hospital (Govt Of
Assam). He did BPT from Srinivas college of
physiotherapy and research Mangalore and MPT in the
specialization of orthopaedics and sport physiotherapy
from Rajiv Gandhi University Of Health Sciences Bangalore in
2012. Presently he is pursuing Ph.D in the same field. He has 4
International Publications. His interested areas of research are,
Musculoskeletal and sport physiotherapy, manual therapy.

Sub154410 frozen shoulder

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